Sarcopenia can also raise the risk of developing cognitive disorders. One study found people with sarcopenia were six times more likely to have cognitive impairments than those without the disease.

We know that exercise can help protect against age-related muscle loss and boost our cognitive health. But what about the role that nutrition plays?

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The power of blended antioxidants

Oxidative stress, which plays a significant role in age-related health issues, is the progressive cellular damage caused by free radicals.

The best defense against oxidative stress? Antioxidants. Consuming foods rich in antioxidants can reduce cell damage and slow down age-related health decline.

In a recent study, researchers gave aged mice a blended antioxidant supplement known as Twendee X (TwX). The supplement contains the following antioxidants:

• Vitamin C
• L-glutamine
• Niacin
• L-cysteine
• Coenzyme Q10 (CoQ10)
• Vitamin B2

Results showed the mice experienced significant improvements in spatial cognition, short-term memory and muscle durability.

These results suggest that such antioxidant cocktails may benefit both memory and muscle strength in aging humans.

The right foods for antioxidants

Many foods are rich in antioxidants, but unfortunately, most of the foods highest in antioxidants are those that most of us don’t eat enough of.

If you’re ready to make an effort to improve your dietary antioxidant intake, look for these:

• Glutamine/l-glutamine: Beef, cabbage, cottage cheese, milk, pork, poultry, raw parsley, raw spinach, ricotta cheese and yogurt
• Niacin (B3): Bananas, beef, beef liver, brown rice, fish, fortified cereals and breads, legumes, nuts, pork, poultry and seeds
• Cysteine/L-cysteine: Beef, egg, poultry and whole grains
• CoQ10: oily fish (such as salmon and tuna), organ meats (such as liver) and whole grains
• Vitamin B2/riboflavin: Almonds, cheese, chicken breast, dairy milk, fortified cereals and breads, eggs, lean beef and pork, organ meats (such as beef liver), salmon, spinach and yogurt
• Vitamin C: bell peppers (all colors), broccoli, Brussels sprouts, cantaloupes, cauliflower, grapefruits, leafy greens (turnip, mustard, beet, collards), honeydew melons, kale, kiwi, lemons, oranges, papayas, snow peas, strawberries, sweet potatoes and tomatoes

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• Vitamin E: Almonds, avocado, leafy greens (beet, mustard, turnip), peanuts, red peppers, spinach (boiled), Swiss chard and sunflower seeds
• Carotenoids (including lutein and zeaxanthin): Apricots, asparagus, beets, broccoli, cantaloupes, carrots, bell peppers, kale, mangos, oranges, peaches, pink grapefruits, pumpkins, winter squash, spinach, sweet potatoes, tangerines, tomatoes, turnip and collard greens and watermelon
• Selenium: Barley, brown rice, Brazil nuts, fish, shellfish, beef and poultry
• Zinc: Beef, cashews, chickpeas, fortified cereals, lentils, oysters, poultry, pumpkin seeds, sesame seeds and shrimp
• Phenolic compounds: Anthocyanins (blueberries, strawberries), catechins (berries, cocoa, tea), coumaric acid (berries, spices), quercetin (apples, onions, red wine), resveratrol (grapes, peanuts, and red wine)

Sources:

Blended antioxidant supplement improves cognition and memory in aged mice — ScienceDaily

A Blended Vitamin Supplement Improves Spatial Cognitive and Short-Term Memory in Aged Mice — International Journal of Molecular Sciences

Antioxidants — Harvard T.H. Chan School of Public Health

Glutamine — Mount Sinai

Niacin – Vitamin B3 — Harvard T.H. Chan School of Public Health

Dietary Cysteine and Other Amino Acids and Stroke Incidence in Women — Stroke

Coenzyme Q10: The essential nutrient — Journal of Pharmacy & BioAllied Sciences

Riboflavin – Vitamin B2 — Harvard T.H. Chan School of Public Health

The post Why antioxidants are the answer for aging brains and muscles appeared first on Easy Health Options®.

In other words, the average level of testosterone for a 45, 55 or 65-year-old man is lower than that of a man of the same age in 2010 — which was lower than that of a man of the same age in 2000!

The question is — why are testosterone levels declining so dramatically? And is there anything you can do to support your testosterone levels?

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Why men’s testosterone levels have dropped

While T levels decline with age, maintaining hormonal balance is more complex. Problems in one area of your health can sneak up and lead to problems in the endocrine system (headquarters for hormonal health).

Three health problems, in particular, have risen to almost epidemic proportions in the U.S. in the past several years, and they all have one thing in common — they can tank testosterone levels…

#1 – Type 2 diabetes

Testosterone is required to help all of the tissues in your body take up blood sugar in response to insulin. It’s no wonder then that men with low T levels are far more likely to have insulin resistance, meaning their bodies have to churn out more insulin to to keep their blood sugar in the normal range.

According to the American Diabetes Association, “If you have Type 2 diabetes, you are twice as likely to suffer from low testosterone as a man without diabetes.”

#2 – Liver problems

Studies show that men with non-alcoholic fatty liver disease, or NAFLD (a condition where there is an excess buildup of triglycerides in the liver), have reduced total testosterone.

Untreated, NAFLD can progress to nonalcoholic steatohepatitis (NASH) and after that, possibly cirrhosis. 75 percent to 90 percent of men with these conditions experience extremely low T.

#3 – Obesity

Obesity is also a driving force behind the low T epidemic. One study showed that 30 percent of overweight men had low T, versus just six percent of those with weight in the normal range

Estrogen dominance can cause abdominal weight gain. That’s because elevated estrogen levels signal the body to reduce testosterone production and store more fat.

Estrogen dominance is a condition that occurs when fake estrogens disrupt hormonal balance and surprisingly can cause health problems for both men and women. In men, low T can impact stamina, muscle tone and prostate health as well as weight gain.

How to fight testosterone theft

While men’s testosterone levels have been under assault for quite a while now, you’re not powerless to do something about it…

First and foremost is to ensure you don’t fall victim to any of the three health conditions that make you a target for low T and declining health.

Many of the steps you can take to improve your health can also improve your testosterone levels, including:

High-intensity interval training and resistance training seem to be especially effective at boosting testosterone.

Eating a high-quality diet of protein, fruits and vegetables and healthy fats is also important (a keto diet has shown real benefits for Low T).

Stress management reduces the stress hormone cortisol which can cause weight gain and lower testosterone.

But you also need to face up to fake estrogens which are almost inescapable in our environment. Luckily, there’s a way to flush them out of your body…

The secret is a compound called Di-Indole Methane, or DIM for short, which is found in cruciferous vegetables like broccoli, cauliflower, and cabbage.

DIM can reduce estrogen levels in the body by stimulating the breakdown of estrogen into beneficial metabolites.

But it takes far more of this little compound than what you can get by sitting down to a dinner packed with veggies.

In fact, it would take a minimum of three pounds of broccoli every day to grab all of DIM’s hormone-supporting benefits. Fortunately, the nutrient is available in supplement form.

Sources:

Why Are Testosterone Levels Decreasing? – Cleveland Clinic

Low Testosterone – American Diabetes Association

8 Major Factors That Cause Low Testosterone Levels – Men’s Health

Low Testosterone (Male Hypogonadism) – Cleveland Clinic

Testosterone in men with advanced liver disease: Abnormalities and implications – Wiley Online Library

Low Testosterone in Males May Warrant Liver Health Assessment and Intervention – NIH

Relevance of low testosterone to non-alcoholic fatty liver disease – NIH

Low Testosterone and Your Health – WebMD

Responses of sex steroid hormones to different intensities of exercise in endurance athletes – The Physiological Society

Endocrinological Roles for Testosterone in Resistance Exercise Responses and Adaptations – NIH

Testosterone-Associated Dietary Pattern Predicts Low Testosterone Levels and Hypogonadism – MDPI

Manipulation of Dietary Intake on Changes in Circulating Testosterone Concentrations – MDPI

Low-fat diets and testosterone in men: Systematic review and meta-analysis of intervention studies – Science Direct

Stress, cortisol, and other appetite-related hormones: Prospective prediction of 6-month changes in food cravings and weight – Wiley Online Journal

The post 3 health conditions that steal your testosterone appeared first on Easy Health Options®.

It’s one of the only carotenoids that accumulates in the retina. When taken with the other (lutein), it works to protect your eyes from the harmful free radicals behind macular degeneration.

But there may be a lot more to zeaxanthin than that. In fact, research indicates this plant-derived compound could help protect against one of our most dreaded diseases…

Could zeaxanthin help against cancer?

Building on years of work by the lab of Dr. Jing Chen of the University of Chicago, researchers led by Chen sought to better understand how nutrients influence the immune system.

Zeaxanthin has already demonstrated anti-inflammatory effects. When screening an extensive blood nutrient library, the team identified zeaxanthin as a compound that directly enhances the activity of a certain type of immune cell (CD8+ T cells) that kills tumor cells. CD8+ T cells rely on a structure known as the T cell receptor (TCR) to recognize and destroy abnormal cells.

Upon interacting with cancer cells, the researchers discovered zeaxanthin stabilizes and strengthens the formation of the TCR complex on CD8+ T cells. This, in turn, triggers more robust intracellular signaling that boosts T cell activation, cytokine production and, importantly, tumor-killing ability.

Then, the researchers tested zeaxanthin in mouse models of cancer. Dietary supplementation with zeaxanthin in these mouse models slowed tumor growth. Plus, when combined with immune checkpoint inhibitors — a type of immunotherapy that has transformed cancer treatment in recent years — zeaxanthin significantly enhanced anti-tumor effects compared to immunotherapy alone.

The researchers extended the findings by testing human T cells engineered to recognize specific tumor antigens. They found that treatment with zeaxanthin improved the ability of these cells to kill melanoma, multiple myeloma, and glioblastoma cells in laboratory experiments.

“Our data show that zeaxanthin improves both natural and engineered T-cell responses, which suggests high translational potential for patients undergoing immunotherapies,” Chen says.

To sum up, the researchers discovered in laboratory and mouse studies that zeaxanthin may strengthen the cancer-fighting activity of immune cells as well as boost the effectiveness of cancer immunotherapy.

Chen says the researchers “were surprised” to uncover zeaxanthin’s new function as an immune booster. “Our study shows that a simple dietary nutrient could complement and strengthen advanced cancer treatments like immunotherapy,” he says.

Zeaxanthin’s many benefits and more to come

Clearly, these results are promising. However, the researchers stress that the work is still at an early stage, with most of the findings coming from laboratory and animal studies.

“Our findings open a new field of nutritional immunology that looks at how specific dietary components interact with the immune system at the molecular level,” Chen says. “With more research, we may discover natural compounds that make today’s cancer therapies more effective and accessible.”

Zeaxanthin is naturally found in vegetables like orange peppers, sweet potatoes, spinach and kale. It’s also available in over-the-counter supplements for eye health, with benefits that go far beyond.

It’s inexpensive, widely available and well-tolerated. It also has a known safety profile, which means it can be safely tested as an adjunct (or addition) to cancer therapies.

Although this research is in its early stages, there really are no downsides to adding zeaxanthin-rich foods or a supplement containing zeaxanthin to your diet today. You’ll be protecting your eye health — and potentially giving yourself a boost against cancer as well.

Sources:

Plant-based nutrient can boost immune cells’ ability to fight cancer — Medical Xpress

Zeaxanthin augments CD8⁺ effector T cell function and immunotherapy efficacy — Cell Reports Medicine

The post The eye vitamin joining the fight against cancer appeared first on Easy Health Options®.

Not only do they increase your risk of developing heart disease and 30 other health conditions and diseases, but they’re also behind the obesity epidemic.

In fact, I wrote about the research that confirmed it’s these foods, not lack of exercise — as previously thought — that’s fueling rising rates of obesity.

But what’s truly disturbing is their impact on reproductive and metabolic health…

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Weight gain is just the beginning

A 2025 study at the University of Copenhagen has confirmed that no matter how little of them you eat, ultra-processed foods lead to weight gain.

Researchers compared the health effects of an unprocessed vs. an ultra-processed diet on the same person, in order to get the most accurate results.

Forty-three men spent three weeks eating each of the two diets, with a cleansing period in between.

The unprocessed and ultra-processed diets had the same amount of calories, protein, carbs, and fats. But half the men were given an extra 500 calories per day.

Regardless of whether those extra 500 calories were involved, men on the ultra-processed diet gained about 1 kg (just over 2 pounds) more fat mass than those on the unprocessed diet.

But it doesn’t end there…

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“Our results prove that ultra-processed foods harm our reproductive and metabolic health, even if they’re not eaten in excess. This indicates that it is the processed nature of these foods that makes them harmful,” says Jessica Preston, lead author of the study.

Men on the ultra-processed diet showed:

• An increase in levels of phthalate cxxMINP, a hormone-disrupting chemical typically found in plastics.
• A decrease in testosterone and follicle-stimulating hormone, both essential for sperm production. In addition to affecting fertility, low sperm count impacts a man’s sex drive and can be a cause of erectile dysfunction.
• An increase in LDL (“bad” cholesterol) levels.
• Altered thyroid hormones.
• Altered markers of systemic inflammation and oxidative stress.

And in case you’ve forgotten, chronic inflammation causes the cellular damage behind a range of diseases from cancer to Parkinson’s disease to diabetes, not to mention an accelerated aging process that makes your body old before its time.

“We were shocked by how many body functions were disrupted by ultra-processed foods, even in healthy young men. The long-term implications are alarming and highlight the need to revise nutritional guidelines to better protect against chronic disease,” says the study’s senior author, Professor Romain Barrès.

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The choice is clear

I hope you don’t need any more convincing.

Whole foods are the best alternatives to ultra-processed foods. But for success, ease into eating more of them and fewer ultra-processed foods.

Ultra-processed foods are made to “hook” you, so don’t blame yourself if it’s hard to let them go. That being said, see about ditching the two ultra-processed foods considered the worst ASAP.

According to Heartfoundation.org, whole foods and minimally processed foods, like these, are what we all should be eating:

• Fresh, frozen and canned vegetables and fruit
• Dried, canned and frozen beans and legumes like lentils and chickpeas
• Whole grains like oats, brown rice, barley and quinoa
• Fresh and frozen poultry and meat
• Fresh, frozen and canned fish and seafood
• Milk and plain yoghurt (try sweetening with honey)
• Eggs
• Nuts and seeds.

An easy way to identify ultra-processed foods is that they are quick, easy, and require little to no preparation — the very definition of “convenience foods.”

They include foods like sweetened cereals, soda and white bread; frozen French fries, chicken nuggets and pizza; processed meats, like hot dogs and bacon; pre-packaged baked goods, like cookies and cakes; and microwaveable meals.

Make the choice today and start taking back your health and your longevity.

Sources:

Why ultra-processed diets make you gain fat even without extra calories — Science Daily

Effect of ultra-processed food consumption on male reproductive and metabolic health — Cell Metabolism

Here’s What Eating Processed Foods for Two Weeks Does to Your Body — Time

Five ways to eat less processed food — Heart Foundation

The post Ultra-processed foods: A metabolic and hormonal disaster for men appeared first on Easy Health Options®.

But there’s another measurement of age that has a far bigger impact…

Known as epigenetic or biological age, it presents a more accurate picture of how well your body’s cells and tissues are functioning. That makes it a more precise measure of age and risk for age-related disease.

The good news is that there are ways to slow down the epigenetic aging process and push back the onset of aging-related diseases, potentially increasing both your healthspan and lifespan.

And research has revealed one of the simplest and most effective ways to reverse your body’s aging clock is something you can do right now…

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Delivering measurable reductions in biological age

In research from Tohoku University, scientists reviewed existing evidence from studies showing that regular exercise, physical activity and fitness may influence epigenetic aging and potentially reverse it.

The results were compelling enough that you may just want to get up and dance — or walk, run, lift weights, go swimming, or engage in any type of exercise that floats your boat.

That’s because the deep dive into study after study revealed the incredible benefits of staying active for reducing epigenetic aging, including:

• A study in mice that demonstrated that endurance and resistance training reduced age-related molecular changes in muscle tissue.
• A human study that found exercising reduced biological age markers in blood and skeletal muscle.
• Research that revealed that sedentary middle-aged women could reduce their epigenetic age by two years after just eight weeks of combined aerobic and strength training.
• Another study that found older men with higher oxygen uptake levels, a key measure of cardiovascular fitness, had significantly slower epigenetic aging.

According to the scientists, “These findings suggest that maintaining physical fitness delays epigenetic aging in multiple organs and supports the notion that exercise as a geroprotector confers benefits to various organs.”

Considering all that can go wrong without exercise, I’m not really surprised.

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Which organs benefit most?

The research also was able to show that while exercise is excellent for slowing aging across your entire body, specific organs grab even bigger benefits.

Obviously, exercise offers benefits for your skeletal muscles, which are used to move your body as you exercise, allowing them to stay strong and healthy.

However, beyond muscle, regular physical training may also slow the aging process in your heart, liver, fat tissue, and even your gut (which controls a significant portion of your immune system).

The scientists do say, though, that if you want to experience the anti-aging benefits of exercise to the fullest extent, the key is to leverage a structured exercise routine that is planned, repetitive, and goal-directed.

That’s because regular exercise, like riding an exercise bike, playing tennis, going for a jog or lifting weights, appears to have more potent effects on slowing epigenetic aging than general physical activity, such as walking or doing household chores.

In addition to exercise, restricting calories can also help reduce aging and even mortality risk. So you have choices. Do what feels right for you.

Sources:

Exercise may actually reverse your body’s aging clock – ScienceDaily

The post Doing this regularly could help you age in reverse appeared first on Easy Health Options®.

You might feel fatigued after a long day of activities or a long night of poor sleep.

But if daily fatigue is your constant companion, the cause may be a condition that’s zapping your energy or a vitamin deficiency — or both.

There’s a large amount of research on the connection between vitamin D and fatigue — particularly, how low vitamin D levels can bring on the debilitating fatigue that is part of a range of conditions — including aging.

For that reason, a group of Italian researchers decided to dig into it and shed light on the mechanisms behind vitamin D’s connections to fatigue…

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The energy vitamin

Vitamin D is not a cure. But a growing body of evidence has emerged indicating the vitamin’s involvement in various processes in the body.

So it’s not surprising that hypovitaminosis D — a deficiency or insufficiency of vitamin D — has been previously linked to heart trouble, neurodegeneration, rheumatoid arthritis and diabetes.

The Italian researchers accessed scientific studies in the databases of PubMed, Scopus, and Web of Science and reviewed factors that play a role in the genesis of fatigue, where the influence of vitamin D has also been demonstrated. 

Here are some of their findings…

Fibromyalgia. There’s no proof of a causal link between low vitamin D levels and the debilitating fatigue of fibromyalgia. Conversely, though, studies have shown a significant improvement in fatigue with higher vitamin D levels.

Autoimmune disorders. Studies offer mixed results regarding whether vitamin D improves fatigue in autoimmune disorders. However, researchers advocate testing their vitamin D levels since low blood levels are frequent among them, and treatment has resulted in a significant decrease in fatigue severity.

Most of the studies on vitamin D and autoimmune conditions have been on reducing the occurrence of autoimmune diseases.

Chronic Fatigue Syndrome (CFS). Most of the research for review on CFS focused on reducing pain and that presented a challenge during the review. However, a meta-analysis revealed low blood levels of vitamin D present in patients with fibromyalgia, and another study clearly demonstrated that correcting the deficiency improved the fatigue symptom.

Multiple Sclerosis (MS). The researchers noted that a good response against fatigue in multiple sclerosis patients when supplemented with vitamin D has been documented.

Aging. This review uncovered a significant body of research supporting a correlation between correcting low levels of vitamin D and the improvement of fatigue in elderly patients. In fact, this correlation was greater than in any of the other conditions examined.

That may be due in part to vitamin D’s role in regulating the development of an anti-aging protein known as klotho. When we’re young, our kidneys produce lots of this protein. But as we age, that production slows.

Klotho exerts anti-aging effects by increasing oxidative stress tolerance and preventing ROS overproduction.

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Can vitamin D help your fatigue?

To sum up the review’s findings, Vitamin D affects fatigue through three main methods:

• it reduces the oxidative stress that’s associated with conditions like chronic fatigue syndrome, fibromyalgia and even aging.
• it regulates fatigue-related neurotransmitters such as serotonin and dopamine.
• it reduces levels of pro-inflammatory cytokines (small proteins that signal the immune system to produce inflammation)

Could vitamin D help fight fatigue levels if you’re relatively healthy? That’s an easy one…

Apparently, a lot of us complain about feelings of fatigue when we see our primary care physicians. So one such group was put to the test.

One hundred and seventy-four adult patients, who presented in a primary care office with fatigue and stable chronic medical conditions completed fatigue assessment questionnaires.

Low vitamin D levels were discovered in 77.2 percent of the patients who complained of fatigue. Those patients received supplement therapy for 5 weeks.

After their levels were normalized, their fatigue symptoms improved significantly!

Your doctor can check your levels if you’re unsure of your D status, but you should know that insufficiency is prevalent in about 45 percent of the adult population. Supplementing vitamin D, or vitamins in general, is effective and safe as long as you don’t take more than directed.

Sources:

Association between vitamin D supplementation and fatigue — News Medical Life Sciences

Vitamin D and Its Role on the Fatigue Mitigation: A Narrative Review — Nutrients

Effect of vitamin D3 on self-perceived fatigue — Pub Med Central

Correction of Low Vitamin D Improves Fatigue: Effect of Correction of Low Vitamin D in Fatigue Study (EViDiF Study) — National Library of Medicine

The post Vitamin D’s ties to autoimmune and age-related fatigue appeared first on Easy Health Options®.

But there is no question that our bodies are left different. Weight gain is easier. Wrinkles appear out of nowhere. Bones become brittle. Cholesterol shoots up.

Having said all that, it is a misconception that postmenopausal women have no influence over our outcomes or our overall sense of well-being.

The previous four blogs dealt with our main pharmacologic option, hormone replacement therapy (HRT). And there is plenty of evidence that HRT would likely be a net positive for many more women than are being offered this intervention today.  If you missed those blogs, you can find them here: Part 1, Part 2, Part 3 and Part 4.  

However, there is much more we can do than just demand to be placed on drugs! We can also help ourselves get through this period and set ourselves up for better health long term through the way we live.

Spoiler alert: If you’re a regular reader of our blogs, you will see a striking similarity to advice I have given many times before…

Turns out, eating right and exercising regularly has a positive impact, regardless of what we are trying to impact positively.

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Mediterranean Diet and Menopausal Symptomatology

The Mediterranean diet, characterized by high consumption of fruits, vegetables, whole grains, legumes, nuts, and olive oil, has been associated with reduced severity of menopausal symptoms.

A study involving more than 6,000 postmenopausal women over nine years found that higher adherence to the Mediterranean Diet correlated with a lower incidence of hot flashes and night sweats.

Phytoestrogens: Plant-Derived Estrogenic Compounds

Phytoestrogens, particularly isoflavones found in soy products, have been investigated for their potential to mitigate menopausal symptoms due to their structural similarity to estradiol.

Randomized studies have shown that phytoestrogen-based treatments may reduce the frequency of hot flashes and night sweats. However, efficacy varies among individuals, possibly due to differences in gut microbiome composition affecting isoflavone metabolism. 

By the way, the idea that phytoestrogens increase breast cancer risk has been summarily debunked. Even women who have gone through breast cancer treatment can enjoy soy products.

Omega-3 Fatty Acids and Mood Regulation

The decline in estrogen during menopause is associated with an increased risk of depression. Omega-3 fatty acids, like those found in Step One Foods, are known to have positive effects on mood, and higher dietary intake of omega-3 fatty acids has been inversely associated with depression prevalence in postmenopausal women.

Micronutrients: Calcium and Vitamin D

Postmenopausal women are at increased risk for osteoporosis, and adequate intake of calcium and vitamin D becomes crucial for bone health. For women over 50, the recommended daily allowance includes 1,200 mg of calcium and 600–800 IU of vitamin D. Individual vitamin D requirements may be higher in order to keep vitamin D levels in the normal range. You can have your vitamin D level assessed through a simple blood test. 

Dietary sources of calcium include dairy products, leafy greens, and fortified foods, while sunlight exposure facilitates endogenous vitamin D synthesis. It is typically difficult to get adequate amounts of vitamin D through diet alone, although eating mushrooms can help.

Exercise: Bone Strength and Beyond

If there was any time in a woman’s life when staying physically active was critical, it’s after menopause. We can take all sorts of calcium supplements, but unless we engage in weight-bearing and resistance training, all that calcium will just go toward making kidney stones. And unless we expend more energy, the slowdown in metabolism most women experience during this time will yield progressive weight gain. The good news is that we don’t need to engage in extreme or exceedingly strenuous routines to see meaningful health gains.

For optimal metabolic health, spending the majority of our time in Zone 2 is ideal. This exercise level helps build aerobic capacity, increases mitochondrial efficiency, burns fat for energy, increases insulin sensitivity, and helps lower blood pressure. Interspersing short, high-intensity intervals is also a good idea to help build cardiovascular fitness.

And how do you know if you’re in Zone 2? To estimate your Zone 2 heart rate range, subtract your age from 220 (this estimates your maximum predicted heart rate), and then multiply that number by 0.6 to get the lower range (60%) of Zone 2, and by 0.7 to get the upper range (70%). You can also know that you’re in Zone 2 by using the talk test: your exercise intensity is low enough that you can carry on a conversation, but high enough that you’re somewhat breathless and can’t sing.

And what about resistance training? Do that at least a couple of times a week for 30 minutes at a time. You can get fancy and work with a trainer at a gym — or make a minor investment in resistance bands and follow a do-it-yourself program at home.

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We Have the Power!

Menopause is a time of tremendous change — physically and psychologically. But it’s definitely not a time to suffer in silence or simply accept our fates.

Helping ourselves through nutrition and physical activity (the EXACT SAME interventions as the ones that yield cholesterol improvements) will go a long way to maximizing overall health outcomes, especially after our estrogen is gone. And, for most women, HRT — whether systemic or local — can be considered a safe, effective and in many ways proactive step to ensuring a smoother transition to the second half of our lives. 

The WHI deprived too many women of quality of life for too long. 

It’s time to move on.

The post Menopause and the big lie: You’ve got choices appeared first on Easy Health Options®.

They were a huge part of the drug culture of the 1960s and were especially known for psychedelic or hallucinogenic effects.

Who would have thought that the same chemical that produced powerful hallucinations might one day be found to help you live a longer and healthier life?

Not me, that’s for sure.

But ongoing research is showing this to be the case.

“Magic mushrooms” might just be the next weapon in the ongoing effort not just to live longer but to ensure those extra years are healthy ones…

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Psilocybin reduces oxidative stress and slows aging

“This study provides strong preclinical evidence that psilocybin may contribute to healthier aging — not just a longer lifespan, but a better quality of life in later years,” says Dr. Ali John Zarrabi, co-investigator of a study at Emory University in Atlanta.

Psilocybin is the active compound found in psychedelic mushrooms. When it is consumed, it forms a metabolite called psilocin.

Emory University researchers dosed 19-month-old mice — an age that in human years would correspond to between 60 and 65 — with psilocybin doses that ranged from 5mg to 15 mg over a 10-month period.

The treated mice lived 30% longer than untreated mice and also experienced better aging — including healthier physical traits like better fur condition and hair regrowth.

Psilocybin is known for its psychological effects, especially in regard to relieving depression and boosting the feel-good hormone, serotonin. But this research indicates that it also targets several key biological markers of aging, including reducing oxidative stress, enhancing DNA mechanisms and maintaining telomere length.

According to the researchers, these underlying cellular processes play a critical role in both aging and disease onset.

How do we explain this?

“Most cells in the body express serotonin receptors, and this study opens a new frontier for how psilocybin could influence systemic aging processes, particularly when administered later in life,” says Louise Hecker, PhD, senior author of the study and former associate professor at Emory University, where the work began and was funded.

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Not a fan of shrooms? You have choices

Dr. Zarrabi emphasized the importance of further research in older adults, as well as the well-documented overlap between physical and mental health.

“My hope is also that if psilocybin-assisted therapy is approved as an intervention for depression by the FDA in 2027, then having a better quality of life would also translate into a longer, healthier life.”

But until then, how do we increase our chances of living a long and healthy life?

For starters, just move. Switching to an active lifestyle at any point in adulthood has been shown to extend lifespan. And you control the dial on effort and benefits. The plan you’ll stick to is the one that will bring you results.

Eat the rainbow. This term was coined for a good reason: Colorful foods are rich in flavonoids that are linked to a disease-free, longer life.

Avoid foods that send aging into overdrive. Certain foods accelerate aging. You’ll find which ones here.

Take advantage of shortcuts that research shows can have a powerful effect on aging, including:

• Coffee: the healthy aging secret under our noses
• The vitamin that slowed biological aging 3 years
• Conquer inflammation, the link between unhealthy aging and a shorter lifespan

Lastly, just in case you need more convincing about the importance of movement, here’s a list of 40 things that can go wrong with your body and health if you don’t exercise at all.

Dr. Jesus Peteiro, the author of a 2019 Spanish study on exercise capacity (how long and how hard you can work out), sums it up for us: “Exercise as much as you can. Fitness protects against death from any cause.”

Sources:

Natural Compound Found in Mushrooms Delays Aging and Extends Lifespan, Study Suggests — SciTech Daily

Psilocybin treatment extends cellular lifespan and improves survival of aged mice — Nature Partner Journals’ Aging

The post The magic mushroom secret for slower aging, longer life appeared first on Easy Health Options®.

But what would be even better is if we could do the same thing for our brains.

After all, the accumulation of damaging amyloid proteins in our brains is what leads to memory problems and puts us on the path to severe cognitive decline.

Now, researchers are discovering how to scrub our brains clean with the power of two natural compounds that, when combined, reverse age-related cellular deficits and improve the brain cells’ ability to clear away damaging buildup.

Here’s what you need to know…

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Energy decline equals protein buildup

According to the researchers, the problems with aging in our brains are due to a lack of energy. That’s probably not a surprise to any of us who have found it more challenging to concentrate and focus with every passing year.

Lead author Gregory Brewer explains that, “As people age, their brains show a decline in neuronal energy levels, which limits the ability to remove unwanted proteins and damaged components.”

A contributor to this buildup is declining levels of guanosine triphosphate, an essential energy molecule for mitochondria — your brain’s energy hubs.

Think of it this way…

Your brain has a natural system that acts like a vacuum cleaner, sucking up and getting rid of amyloid proteins. But it has to have the energy available to power that vacuum.

The solution? Reinstate youthful levels of guanosine triphosphate — and that’s what Brewer and his team are working on…

The supplements that provide the solution

They discovered that a combination of naturally occurring compounds — nicotinamide (a form of vitamin B3) and epigallocatechin gallate (a green tea antioxidant) — restored brain energy levels in aged neurons to those typically seen in younger cells in just 24 hours.

And this revival triggered a cascade of benefits, including improved energy metabolism and efficient clearance of amyloid beta aggregates — hallmarks of Alzheimer’s disease.

According to Brewer, “We found that restoring energy levels helps neurons regain this critical cleanup function.”

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To top it off, restoring brain energy levels also reduced oxidative stress, which leads to neurodegeneration in the brain.

Past research has shown that limiting oxidative stress can reduce the damage typically caused by beta-amyloid.

As Brewer points out, “By supplementing the brain’s energy systems with compounds that are already available as dietary supplements, we may have a new path toward treating age-related cognitive decline and Alzheimer’s disease.”

It seems then that the one-two supplement punch of nicotinamide and epigallocatechin gallate could be the start to revving up mitochondrial energy in the brain and cleaning out cognitive decline for better brain health — no matter what your age.

However, we’re not there yet…

He cautioned, “More work is going to be required to find the best way to administer this treatment, since a recent clinical trial involving UC Irvine researchers showed that oral nicotinamide was not very effective because of inactivation in the bloodstream.”

Until then, you can read here about how to start supporting your power-generating mitochondria today.

Sources:

UC Irvine researchers find combination of natural compounds for brain cleaning — EurekAlert!

The post Researchers find ‘cleaning combo’ clears brain buildup appeared first on Easy Health Options®.

But what if it’s really more akin to an infection?

That’s what researchers say advanced genetic sequencing science is revealing — along with potential treatments capable of targeting the protein behind senescence (the process of deterioration) to stop aging’s “spread.”

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Aging’s dirty little secret

Past research has shown that aging isn’t limited to one area of the body. Instead, when it starts, it begins to diffuse throughout the body.

Sort of like an infection…

One day, usually after the age of 40, you may start feeling arthritis-like issues in, say, your knee. By the following year, not only is your knee aching and stiff, but your back, neck and other areas have joined in.

Before you know it, each year you’re suffering from another sign of aging in another part of your body. But your best friend seems to be doing just fine.

While researchers have been well aware of the spreading effects of aging throughout the body, the mechanisms of how exactly it happens haven’t been so easy to nail down.

Cue a team of researchers from Korea University College of Medicine…

They were able to prove that cellular aging (a process known as senescence) begins due to a protein known as HMGB1 — we’ll call it “aging’s dirty little secret” — that potentially accelerates the aging process as it spreads from one part of the body to another.

They also may have found a way to stop it…

Sending out the aging signal

Their research, published in the journal Metabolism, revealed that this protein acts as an “aging messenger” that carries signals from cell to cell, transforming otherwise healthy cells into aged and dysfunctional ones.

Here’s how it works…

When HMGB1 is in an oxygen-rich environment, it functions normally. However, when there is a lack of oxygen in the cellular environment that surrounds the protein, it becomes reduced — and that’s when things go bad.

HMGB1 then binds to cellular receptors, activating the JAK/STAT and NF-κB pathways in the cells, which are known to promote inflammation and cue the cells to age.

This triggers a molecular signaling cascade: Aging cells produce more HMGB1, which spreads to other cells, activating their senescence pathways as well — in effect causing aging to spread like a virus from cell to cell.

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Putting an aging ‘do not disturb’ sign on your cells

The good news in all of this is that when the scientists treated mice with antibodies that block HMGB1 from activating the JAK/STAT pathway, they were able to reduce age-related dysfunction.

What does that mean for us?

Well, they hope that in the future, a drug treatment can be developed using a class of medications, known as JAK2 inhibitors, to essentially keep HMGB1 from activating the pathways it used to spread its dirty work — otherwise known as aging.

I’m excited about that news — but even more excited about all the natural compounds already known to be JAK2 inhibitors. And many of them are already compounds associated with healthy aging.

I’m sure you’ll recognize a few of these, and may already be up to speed on their anti-aging reputations:

• Resveratrol –A potent antioxidant found in the skin of red grapes, and responsible for widespread interest in the field of anti-aging research, made popular by Dr. David Sinclair and colleagues in the early 2000s.
• Curcumin – The active component of turmeric that offers potential anti-aging effects due to its antioxidant and anti-inflammatory properties that may combat age-related disease and promote healthy aging.
• Bergamottin –  A natural antioxidant phenol found in the peel of the bergamot orange, grapefruit and lemon, with anti-senescence, anti-aging and immune-modulating effects.
• Garcinol –  Extracted from Garcinia indica fruit, peel and leaves, and is a powerful antioxidant.
• Quercetin – A flavonoid found in fruits and vegetables that shows promise for anti-aging due to its antioxidant, anti-inflammatory and senolytic properties.

They won’t stop aging, but I have my doubts that any drug would ever be able to do that either. But they can certainly help you age more healthily.

Sources:

Aging Spreads Through The Body Like An Infection, And This Protein Could Be To Blame — Study Finds

Propagation of senescent phenotypes by extracellular HMGB1 is dependent on its redox state — Metabolism

The Role of Natural Products as Inhibitors of JAK/STAT Signaling Pathways in Glioblastoma Treatment — NIH

Quercetin as a JAK–STAT inhibitor: a potential role in solid tumors and neurodegenerative diseases — BMC

The post The protein that causes aging to spread like an infection appeared first on Easy Health Options®.

Nearly 1 in 4 American adults don’t get the weekly two days of muscle training and 150 minutes of moderate exercise recommended by the Centers for Disease Control and Prevention. And only 1 out of every 10 adults knows how much and what kinds of exercise they need to get to protect against health ailments.

It’s having a big impact on our health. Physical inactivity is at the root of an estimated 3.2 million deaths every year.

If you are one of those who mean to get regular exercise but never quite manage it, today may be a good day to start — especially if you want to live longer….

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It’s never too late to move more

An international team of researchers sought to find out if differing patterns of physical activity, as well as its cumulative impact during adulthood, might be linked with a lower risk of death from all causes, as well as specifically from cardiovascular disease and cancer.

They dug into research databases for relevant studies that assessed physical activity at two or more points in time. Their review ultimately included 85 studies published in English up to April 2024, with sample sizes ranging from 357 to 6,572,984 participants. To overcome the challenges of different analytical methods, the researchers carried out separate analyses for each study.

Overall, pooled data analysis of the study results showed that a higher level of physical activity was connected with lower risks of all the included outcomes.

When looking deeper, the results were even more impressive. Consistently active people had an estimated 30-40 percent lower risk of dying from any cause. And those who increased their levels of activity from below those recommended for good health had a 20-25 percent lower risk of death from any cause.

More specifically, participants who switched from being physically inactive to being more active were 22 percent less likely to die from any cause than those who remained inactive. In comparison, those who increased their leisure-time physical activity levels had a 27 percent lower risk of all-cause death.

As expected, swapping an active lifestyle for an inactive one wasn’t associated with a lower risk of death from any cause.

The bottom line: switching to a more active lifestyle at any point in adulthood may extend lifespan, and it’s never too late to start.

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Cardiovascular risk was the big winner

Generally, the associations between a high level of physical activity and a lower risk of death were more evident for cardiovascular disease than for cancer, though both showed benefits.

Compared with consistently inactive participants, those who were consistently active overall or were active only in their leisure time were around 40 percent less likely to die from cardiovascular disease and 25 percent less likely to die from cancer.

Despite these findings, in general, the evidence for the connections between physical activity patterns and death from a specific cause remained inconclusive, especially for cancer death.

According to the pooled data, people who were consistently active or who became active had lower risks of death from any cause, and specifically death from cardiovascular disease, when they met the recommended weekly physical activity levels.

However, those who were consistently physically active and performing more than the recommended maximum weekly amount of moderate to vigorous intensity exercise only saw a slight additional reduction in risk.

In other words, don’t sweat it if you can’t go harder.

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The researchers note that maintaining or increasing physical activity at levels below the recommended amount was linked with appreciable health benefits. This suggests that even minimal physical activity is better than none, they say.

Despite the limitations, including the subjective nature of the physical activity assessments, the researchers say the findings have significant health implications for starting and maintaining regular exercise.

So what does this mean for you? If you’re already exercising regularly, keep it up. And if you’re not, try easing into an exercise regimen by adding a brisk 30-minute morning or evening walk to your daily routine at least a few times a week. Walking doesn’t require a gym, and the only equipment you’ll need is a pair of sneakers.

If motivation is a problem, do what my husband does and take your phone with you so you can listen to music, an audiobook or a podcast while you walk. It really helps give you something to look forward to.

Sources:

It’s never too late: Just moving more could add years to your life — ScienceDaily

Physical activity trajectories and accumulation over adulthood and their associations with all-cause and cause-specific mortality: a systematic review and meta-analysis — British Journal of Sports Medicine

The Truth About Exercising and Aging — WebMD

How physical inactivity impacts you as you age — Northwell Health

The post Just ‘move more’ to lower disease and lengthen lifespan appeared first on Easy Health Options®.

But what if you pop a multivitamin or have a bowl of cereal fortified with vitamins and minerals every day? Turns out, many of those supplements and breakfast cereals don’t have nearly enough of the daily nutrients you need.

In fact, research shows there are five specific nutrients most Americans aren’t getting enough of from their diet…

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The 5 nutrients we need the most

A healthy body needs a variety of nutrients, including carbohydrates, fats, proteins, water and vitamins and minerals. Carlene Starck, a nutrition scientist and biochemist at the nutrition research and communications company FOODiQ Global, tells ScienceNews Americans’ diets often have tons of high-energy foods but are light on other essential nutrients.

“We know that what we eat affects the prevalence of cardiovascular disease, type 2 diabetes and cancer,” Starck says.

An Australian team led by Starck analyzed data from the U.S. National Health and Nutrition Examination Survey (NHANES) on people’s intake of 24 essential nutrients. It identified where the participants weren’t meeting recommendations. The analysis also took into account the health benefits of each nutrient based on scientific evidence.

Results narrowed the list to five priority nutrients Americans need to get more of: calcium, fiber, magnesium, and vitamins D and E. Optimal amounts of these nutrients are key for disease prevention. For instance:

• Calcium protects bone health and could help prevent osteoporosis.
• Fiber helps control blood sugar and can lower your risk of diabetes and heart disease.
• Magnesium improves bone health, regulates blood sugar, balances blood pressure, plays a role in heart rhythm and supports nerve and muscle function.
• Vitamin D supports bone health and can lower your risk of certain types of cancer and autoimmune diseases.
• Vitamin E has antioxidant and anti-inflammatory properties and bolsters your immune system.

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No nutritional ‘magic bullet’

Because the nutrients span different food groups, no one food will give people everything they need, Starck says. “There’s no superfood; there’s no magic bullet,” she tells ScienceNews.

And that includes supplements, according to Sander Kersten, a nutrition scientist at Cornell University. For example, several studies of supplemental vitamins D and E haven’t shown clear benefits.

Instead of focusing on individual nutrient levels, both Kersten and Starck tell ScienceNews people should think more holistically.

“We need nutrients, but ultimately we eat foods,” Kersten says. The results of the study can point to foods that make our diets better, he adds.

Starck recommends thinking in terms of meals that pull a few key nutrients together at one time, like avocado and salmon on whole-grain toast. Avocado has plenty of vitamin E and fiber. At the same time, salmon is rich in vitamin D. The whole-grain toast contributes additional fiber, and all three ingredients add magnesium to the mix.

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The diet that covers all the bases

One thing is clear from this research: supplements are no substitute for a diet rich in fruits, vegetables, whole grains and lean protein. One diet that covers all these bases is the Mediterranean diet, which has plenty of foods that contain one or more of the five nutrients mentioned above.

Servings vary by food group, but generally include:

• 2-3 servings of fruit and 4 or more servings of vegetables per day.
• 3 or more servings of legumes or beans per week.
• 4 servings of nuts or seeds per week (1/4 cup equals one serving). 
• Grains, including whole grains, should be consumed 3 times daily.
• Fish and seafood: 3 or more servings per week.
• Replace butter and refined oils with extra virgin olive oil. 

But, as good as the Mediterranean diet is, it’s not perfect…

One nutrient that’s a little tougher to get from the largely plant-based diet is calcium. Leafy greens like spinach contain calcium, but because your body has a hard time absorbing the calcium from greens, you would have to eat a bucketful to get your daily recommended dose.

The solution? Add some low-fat dairy, like plain yogurt mixed with fresh fruit, for breakfast or a snack. One serving of yogurt contains nearly double the calcium of spinach, and your body absorbs that calcium more easily.

If you need to supplement your calcium, stick to the RDA, and remember the importance of vitamin D3 to boost absorption and K2 to get it to your bones.

Sources:

These 5 nutrients might be lacking in your diet — ScienceNews

Identification of Priority Nutrients in the US: Targeting Malnutrition to Address Diet-Related Disease Across the Lifespan — Nutrients

Food Sources of Calcium — Dietary Guidelines for Americans

Calcium: Fact Sheet for Health Professionals — National Institutes of Health Office of Dietary Supplements

The post 5 critical nutrients Americans fall short on appeared first on Easy Health Options®.

So, when I heard that a particular compound already known for supporting healthy blood vessels and blood pressure was making waves in anti-aging skincare — I had to tell you about it.

Pterostilbene is an antioxidant produced by plants to protect themselves against viruses, bacteria and fungi. It’s found in abundance in blueberries, and it’s a cousin to resveratrol.

You may already know about the anti-aging potential of resveratrol. It enhances skin elasticity by stimulating collagen production and protecting against oxidative damage.

But here’s why pterostilbene may have resveratrol beat…

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Significant breakthrough in just 28 days

One area where pterostilbene outperforms resveratrol is in “bioavailability” — a term that refers to the amount of a substance that enters the bloodstream after it’s introduced into the body and can have an active effect.

Resveratrol’s bioavailability is about one percent (though pairing it with grape seed extract can improve that significantly). But from the get-go, pterostilbene’s bioavailability is estimated to be around 80-95%!

When pterostilbene is consumed from foods or supplements, it’s absorbed through the stomach lining and cell membranes to reach the bloodstream. But what happens when it’s applied to our skin?

To find out, researchers in China had participants apply an emulsion containing it on one side of their face and a control cream on the other.

After just 28 days, the scientists had this to say: “Our results indicated that the pterostilbene emulsion remarkably improved skin elasticity, firmness, and reduced wrinkles, such as forehead, undereye, and Crow’s feet wrinkles.”

It also:

• Increased the thickness of the epidermis layer
• Enhanced collagen and elastic fibers
• And minimized skin pores

In fact, the improvements seen in the skin were so significant that the researchers say pterostilbene “may change the way we approach anti-aging skincare formulations.”

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From youthful skin to youthful blood vessels

If your goal is healthy, younger-looking skin, leveraging the power of blueberries to turn back the hands of time could be the key.

But that’s not the only benefit you can expect…

As I mentioned earlier, pterostilbene is also known for promoting healthy blood pressure. Here are just a few of the mechanisms of action supported by research:

Antioxidant activity. Oxidative stress interferes with the normal function of the endothelial cells that line artery walls. By inhibiting damage induced by oxidative stress, pterostilbene helps alleviate hypertension.

Anti-inflammatory activity. Pterostilbene can successfully reverse the elevation of pro-inflammatory cytokines. Not only does this help relieve inflammation of the endothelial lining of the arteries, but it also exerts anti-atherosclerotic activity. Atherosclerosis is a condition that can age you before you even know you have it.

And that’s the connection between skin aging and blood pressure — youthful arteries.

Blood vessels harden with age, poor habits and exposure to environmental toxins, oxidative stress and inflammation. That results in loss of elasticity and changes in blood pressure. And when blood vessels lose their firmness, the walls of your arteries become prone to injury.

Those same saboteurs and those same changes to the tissue that make up your skin contribute to visible signs of aging.

So, from the inside out, it makes sense to include pterostilbene in your anti-aging routine, not only to help maintain the youthfulness of your skin, but of your blood vessels as well!

Sources:

Natural Molecule Shows Remarkable Anti-Aging Results After Just 28 Days — SciTechDaily

Bioavailability of resveratrol — Annals of the New York Academy of Science

Effects of Pterostilbene on Cardiovascular Health and Disease — Current Issues in Molecular Biology

The post The breakthrough that reversed skin aging in 28 days appeared first on Easy Health Options®.

Maybe it’s because women cross a proverbial threshold — menstruation — that eventually opens a door for young women to begin regular gynecological checkups. Along with those yearly visits come screenings for female-specific cancers.

This kickstarts a habit of regular preventive care that becomes ingrained. But for men, it’s a different story…

Our culture gives men the message that they need to “be strong” and “handle things.”

This message makes a lot of men — especially young men — reluctant to take care of their health, and it’s a habit that’s slow to change.

A new study shows just how big an impact this is having on the health of men, and the news isn’t good…

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An ounce of prevention = a pound of cure

It’s not just a saying. Preventing disease really is a much easier route than curing one. But it’s not a message men have gotten — and it shows…

Last year, Harvard researchers shared that the life expectancy gap between men and women had grown to as much as six years. Sadly, experts say it’s due in part to preventable deaths.

“When you’re in your 20s, 30s, you’re indestructible, you’re Superman,” says Scott Stephens, a 62-year-old who never thought about his health care until he was in his 40s and his father developed prostate cancer.

“It’s also a badge of courage; why should I go? I don’t need to call my doctor.”

A new survey by the Cleveland Clinic looked at members of Gen Z (born 1997-2005), Millennials (born 1981-1996), Gen X (born 1965-1980) and Boomers+ (born before 1965).

They grouped Gen X and Boomers together and Millennials and Gen Z together. Then they compared their survey answers to some specific questions:

• Get a yearly physical (61% Gen X and Boomers+ vs. 32% Millennials and Gen Z men)
• Avoid smoking/vaping (60% Gen X and Boomers+ vs. 43% Millennials and Gen Z men)
• Address mental health (53% Gen X and Boomers+ 59% Millennials and Gen Z men)

Additional revelations from the survey were:

• Most men fear getting cancer (74%), but rates of screenings and cancer awareness remain low.
• One-third of Gen X and Boomers+ who are eligible for colorectal cancer screening have not been screened or are not sure if they’ve been screened.
• A quarter of men with an average risk of prostate cancer (50+) have not been screened or are not sure if they’ve been screened.

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What men should do to protect their health

There’s a clear generational divide in how men approach health. But if we jumped back in time 30 years or so, we’d likely find the exact same habits playing out.

In other words, Gen Z isn’t the first generation of young men to place a low priority on their health. It seems that it’s just a matter of getting older before men begin to take health seriously — when symptoms crop up that require attention.

But regardless of age, there are steps men should be taking at each stage of life to safeguard their health:

In your 20s or 30s. Stop relying on the Internet. Make an appointment and get a handle on how healthy (or unhealthy) you are.

“Men should first come around age 21 for general health maintenance,” says Dr. Messmer. It’s also a good time to start measuring cholesterol, especially if there’s a family history of heart disease.

In your 40s. Colon cancer screening should start when men are in their 40s and then repeat every 10 years. If you have a family history, screening should start five years earlier than the age at which the relative was diagnosed and be repeated every five years.

Prostate, bladder, and kidney cancer screening should also begin in a man’s 40s.

In your 50s or 60s. This is the time when urologic issues tend to crop up. Many men come in with erectile dysfunction but discover they have a more serious prostate problem. Besides the standard PSA test, doctors now have other ways to gauge a man’s risk.

Finally, Dr. Ryon McDermott of the University of South Alabama points out, “The brain and the body are obviously very connected.” It’s never too late to start taking care of your mental health.

Sources:

National Cleveland Clinic Survey Examines Generational Divide in Men’s Health — Cleveland Clinic

It’s Time to Man Up and See a Doctor — Web MD

Widening Gender Gap in Life Expectancy in the US, 2010-2021 — JAMA Internal Medicine

Mars vs. Venus: The gender gap in health — Harvard Medical School

The post The unnecessary reason for men’s increasingly shorter lifespans appeared first on Easy Health Options®.

But the most compelling reason has to do with one special nutrient…

Unlike fish oil, krill oil also contains astaxanthin, a powerful antioxidant that’s many times more potent than vitamin C, beta-carotene and vitamin E.

Astaxanthin not only pumps up the omega-3 benefits of krill oil, it gives the body’s aging processes quite a challenge, in part because of its powerful impacts on the body’s most vital systems and organs.

So let’s dive into what this amazing antioxidant is, where it comes from and what the research says about it…

What exactly is astaxanthin?

Astaxanthin is a red-colored ketocarotenoid first identified and isolated in 1938 by scientist Richard Kuhn, who discovered it while studying lobsters to find out what gave them their color. Kuhn won the 1938 Nobel Prize in Chemistry for his work on carotenoids and vitamins.

Astaxanthin is found mainly in the algae Haematococcus pluvialis and the yeast Xanthophyllomyces dendrorhous, both of which produce it naturally. The algae are a food source for a number of sea creatures, and astaxanthin is what turns their shells and flesh pink.

Because krill feed almost exclusively on these algae, they contain higher amounts of astaxanthin than other marine animals like shrimp, salmon, trout and lobster.

How astaxanthin works in the body

As a carotenoid, astaxanthin is fat-soluble and has antioxidant properties that help protect your cells from free radicals and oxidative stress.

Astaxanthin acts by neutralizing reactive oxygen species (ROS) on the inner and outer layers of cell membranes. This keeps ROS from doing damage to the basic building blocks of the cell including DNA, protein and lipids.

Here are some specific ways astaxanthin supports your overall health….

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Astaxanthin and inflammation

Like many antioxidants, astaxanthin may help to reduce inflammation. But astaxanthin’s specific ability to neutralize ROS helps reduce proteins that can cause inflammatory diseases such as celiac disease, rheumatoid arthritis, heart disease and diabetes.

Not only does astaxanthin fight inflammation, it also assists our mitochondria, the energy powerhouses located in our cells. Having mitochondria functioning at their best gives our bodies optimal energy and helps it operate at peak performance.

By reducing inflammation, astaxanthin supports improved mitochondrial performance, as well as providing antioxidant support to ward off free radical damage.

Studies show astaxanthin’s anti-inflammatory capabilities can help with exercise recovery by protecting the body from overproduction of free radicals, inhibiting the production of excess lactic acid in the muscles, reducing fatigue and improving muscle strength.

Astaxanthin and immune system health

Along with fighting inflammation, astaxanthin can boost your immune system by helping to activate white blood cells known as T-cells as well as natural killer cells. T-cells attack malicious cells based on antigen markers, while natural killer cells work even faster to stop foreign invaders from harming your health.

But as important as it is for an immune system to be reactive — it’s just as important not to be overactive to avoid an autoimmune response. Astaxanthin’s anti-inflammatory properties help modulate a balanced immune response.

Astaxanthin and brain health

Carotenoids like astaxanthin help support good brain health by reducing the risks for neurodegenerative conditions like Alzheimer’s disease, Parkinson’s disease, amyotrophic lateral sclerosis and cognitive impairment.

But one advantage astaxanthin has is that its molecular structure is small enough to allow it to cross the blood-brain barrier. This gives it added ability to protect the brain and possibly even slow the onset rate of cognitive disorders.

Astaxanthin may also literally grow your brain. According to one study, astaxanthin can promote the growth of new brain cells in the hippocampus, the area of the brain responsible for learning and memory. Researchers also found that those new cells had even greater learning and memory capacity.

Astaxanthin and heart health

Like omega-3s, astaxanthin has great benefits for your heart, including blood pressure. In one double-blind placebo-controlled study, postmenopausal women who took astaxanthin experienced a 7 percent reduction in systolic blood pressure (the top number) and a 4 percent reduction in diastolic pressure (the bottom number).

Astaxanthin can also help reduce LDL, the “bad” kind of cholesterol, and raise HDL, or “good” cholesterol. And since astaxanthin works in tandem with omega-3s in krill oil, you get a greater cholesterol-lowering benefit than with regular fish oil.

Results of one study showed patients who took 1 to 1.5 grams of krill oil a day showed a significantly higher decrease in bad cholesterol than patients who took three times that dose in fish oil.

Astaxanthin and blood sugar problems

Astaxanthin could be useful in preventing diabetic nephropathy, a disorder of the kidneys. According to one study in mice, the antioxidant activity of astaxanthin helped lower blood sugar, reduce stress on the kidneys and prevent renal cell damage.

Another study showed astaxanthin protected cells against the oxidative damage caused by high blood sugar levels. This damage can cause several complications in diabetics, including kidney disease, nerve damage (neuropathy) and vision problems (retinopathy).

Astaxanthin and eye health

While chemically astaxanthin shares some similarities with other carotenoids (like beta-carotene), it’s more closely related to oxygenated xanthophylls like lutein and zeaxanthin, both known for improving eye health. And like these two nutrients, astaxanthin can also help protect your vision.

But here astaxanthin has an extra edge, like it does in the brain…

Not only can the antioxidant cross the blood-brain barrier, but the same process holds for the retinal barrier.

In studies of patients with age-related macular degeneration (AMD), supplementing astaxanthin and other carotenoids were found to significantly improve retinal electrical outputs, which helps slow AMD damage.

Glaucoma involves an increase in the pressure of fluid inside the eyeball that causes oxidant damage and loss of blood flow and eventually results in retinal cell death. In eyes with experimentally induced glaucoma, astaxanthin restored these retinal parameters to normal.

Another study showed that astaxanthin supplementation resulted in a 46 percent reduction of eye strain caused by visual display terminals.

Astaxanthin and skin health

Astaxanthin appears to act as a sort of natural internal sunscreen by helping to protect your skin from damage by the sun’s ultraviolet (UV) rays. The nutrient builds up in the top two layers of skin, helping to block UV penetration and reduce existing sun damage like wrinkles and lack of elasticity.

Astaxanthin and longevity

Researchers have found that astaxanthin can significantly increase the activation of the so-called “longevity gene” located in our heart tissue. In fact, animals in one study that were fed large amounts of astaxanthin had a 90 percent increase in the activation of this gene.

Healthy aging is a hot topic in discussions surrounding lifespan. This is the intersection referred to as healthspan. Numerous studies have shown that supplementing astaxanthin appears most beneficial for many of the aging processes and organs most challenged by aging including the brain, eyes and skin, not to mention energy levels.

To supplement or not to supplement

There’s no doubt that seafood is a rich source of astaxanthin — but it has to be the right kind. Wild salmon contains between 26 and 38 mg of astaxanthin per kilogram, while farmed salmon only contains 6 to 8 mg per kilogram.

Some experts recommend getting about 3.8 mg of astaxanthin a day, the amount found in roughly 5.8 ounces of salmon. However, others say a therapeutic dose of astaxanthin is anywhere from 4 to 12 milligrams a day, which makes it difficult to get through diet alone.

That’s why supplementing with astaxanthin-rich krill oil is a great alternative. Just remember, because it’s a fat-soluble carotenoid, take it after a meal for best absorption in the body.

Sources:

Health Benefits of Astaxanthin — Nourish by WebMD

Health effects of astaxanthin have been long studied — Natural Products Insider

Beneficial and Detrimental Effects of Reactive Oxygen Species on Lifespan: A Comprehensive Review of Comparative and Experimental Studies — Frontiers in Cell and Developmental Biology

Biological and neurological activities of astaxanthin (Review) — Molecular Medicine Report

Astaxanthin: Sources, Extraction, Stability, Biological Activities and Its Commercial Applications—A Review — Marine Drugs

Clinical Applications of Astaxanthin in the Treatment of Ocular Diseases: Emerging Insights — Marine Drugs

The Microalgae-Sourced  Carotenoid That Delivers Broad Spectrum Antiaging Benefits — Clinical Education

The post Astaxanthin: The antioxidant that challenges aging appeared first on Easy Health Options®.

Given that this is a normal physiologic process and not a disease, menopause is supposed to be something women just endure and move beyond. But if you’ve been through it yourself or are in the midst of “the change,” you’ve probably realized that nothing about this process feels even remotely normal!

That is, unless you think hot flashes, night sweats, mood changes, sleep disturbance, vaginal dryness, urinary urgency and incontinence, weight gain, decreased libido, thinning hair and skin, memory and concentration problems, joint and muscle pain — not to mention heart palpitations and out-of-nowhere high LDL — are all normal!

I don’t typically venture outside of conditions that I manage. I’m a cardiologist. I deal with chest pain and EKGs. But I felt compelled to write this blog series because I continue to see so many female patients suffering with many of the symptoms related to menopause while being denied — or fearful of — a proven life-enhancing intervention. All based on faulty data.

I’m talking about hormone replacement therapy (HRT).

You’ve probably heard the warnings: It causes breast cancer. It’s dangerous. It does more harm than good. These statements have become medical dogma. But they are outdated, oversimplified, and in many cases — flat-out wrong.

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Why HRT Deserves a Second Look

If you’re a woman navigating perimenopause or menopause, what you’ve been told about HRT — or what you think you know about HRT — could be standing between you and a dramatically better life.

This blog series is meant to give you the knowledge you need to not only help yourself through this time (and beyond) but also understand a treatment option that is out there. So that you can have a more informed conversation with your care provider.

To be clear — I am NOT advocating that every woman should go on HRT. But every woman should at least be given the opportunity to determine for themselves if it is right for them.

The Whole-Body Impact of Sex Hormones

First, here’s something really important you need to understand: Sex hormones (estrogen, progesterone, testosterone) don’t just have roles in reproduction and sex drive. Receptors for these hormones exist all over our bodies! Not surprisingly, menopause-related whole-body reductions in sex hormone levels have far-reaching effects.

Hormones and Bladder Health

One organ impacted by those far-reaching effects is the bladder. It happens to be very hormone-sensitive. And not just in terms of maintaining urinary continence. When estrogen levels decline, the microbiome of the bladder and vagina changes, making postmenopausal women far more susceptible to urinary tract infections. Indeed, about 20% of women over age 65 will develop a UTI, with close to 1/3 of these experiencing recurrent infections.

Hormones and Bone Loss

The skeletal system is a major casualty. In the absence of estrogen, women can lose as much as 20% of their bone density in the first 5 years post-menopause, with bone loss slowing down to about 1% per year thereafter. As a result, one in two women over age 50 will experience an osteoporosis-related bone fracture. This is incredibly serious — a hip fracture can be a terminal event. Close to 30% of hip fracture patients will not survive one year. And even if a woman makes it past that, the odds of her getting back to prior levels of activity are low.

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Hormones and Brain Function

Brain? The risk of developing dementia and Alzheimer’s is significantly higher for women. The role of sex hormones is not fully worked out here, but even in the shorter term, sleep disturbance can become a huge issue affecting mental clarity.  Estrogen and progesterone help drive circadian rhythms, and the wild fluctuations in their levels during perimenopause can do a number on the sleep cycle. Plus, it’s hard to sleep when you’re drenched in sweat.

Hormones and Heart Disease

As a cardiologist, I see this in my practice almost daily. Heart rhythm disturbances become more common around menopause, probably due to a combination of factors — especially sleep disturbance. Fortunately, most of these rhythm issues are benign. But that doesn’t mean they feel good! Cholesterol profiles almost universally get worse as LDL receptors become less active. As a result heart disease risk accelerates rapidly in postmenopausal women, reaching par with men of equal age about 10 years post our hormonal upheaval.

Menopause-Related Impact on Life Quality

Add vaginal dryness and loss of libido and you have a marked decline in quality of life — right around the halfway point in women’s lifespans.

These may all be “natural” consequences of changes in hormone levels, but are we just supposed to sit back and accept all that?

Would MEN accept all that??

This is part one of a five-part series. See:

Menopause and the Big Lie , Part 1

Menopuase and the Big Lie, Part 2

Menopause and the big lie: The facts they left out, Part 3

Menopause and the big lie: The lasting impact, Part 4

The post Menopause and the big lie appeared first on Easy Health Options®.

No matter what you call it, most of us have experienced those unsettling feelings of dread and anxiety in anticipation of the beginning of another long, busy, likely stressful week.

Even though the feelings may intensify Monday morning, we march on, able to dismiss them — until Sunday night rolls around again.

Unfortunately, it’s a cycle that evidence connects to dangerous physiological processes…

One meta-analysis of data from population statistics found a 19 percent increase in the incidence of confirmed heart attacks and sudden cardiac deaths on Mondays.

Now, another study appears to validate the biological components associated with Monday-related stress…

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Mondays acts as a ‘stress amplifier’

Researchers led by Tarani Chandola of the University of Hong Kong (HKU) wanted to determine whether the association between anxiety and dysregulation of the hypothalamic-pituitary-adrenal (HPA) axis — the body’s central stress response system — was greater on Mondays.

The HPA axis regulates stress hormones, such as cortisol, which can contribute to hypertension, insulin resistance, and immune system dysfunction if they’re chronically elevated.

The researchers took a look at 3,511 adults aged 50 and older living in England who participated in the English Longitudinal Study of Ageing (ELSA). The participants submitted hair samples and were asked about their anxiety levels and on what days they experienced anxiety.

The results were striking. Participants who felt anxious on Mondays showed significantly higher long-term stress hormone levels up to two months later. Their hair samples showed a 23 percent higher level of cortisol compared to peers who were anxious on other days of the week.

What’s more, this “anxious Monday” effect was observed in both working and retired participants. This indicates a deep-seated link between the start of the week and dysregulation of the body’s stress response, one that lingers well after retirement.

Prior research has observed higher cortisol levels on weekdays versus weekends. But this is the first study to single out Mondays as particularly disruptive.

“Mondays act as a cultural ‘stress amplifier,’” Chandola says. “For some older adults, the week’s transition triggers a biological cascade that lingers for months. This isn’t about work — it’s about how deeply ingrained Mondays are in our stress physiology, even after careers end.”

About 75 percent of the Monday effect was due to the greater impact of feeling anxious on Mondays compared to other days.

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Ways to battle ‘anxious Mondays’

The study emphasizes how the anxiety around Mondays can become biologically embedded in our bodies, with chronic stress hormone dysregulation posing long-term cardiovascular risks.

By addressing the stress specific to Mondays, the researchers hope to unlock new strategies to combat heart disease in aging populations.

“The cumulative life course effects of feeling anxious on Monday, to which people do not adapt, suggests the need to identify why some people do not adapt to the Monday effect, while others are more resilient,” the researchers write in the study.

One reason some people may handle their Monday stress better is that they have a system in place to address the beginning of the week. If you find yourself battling the “Sunday scaries,” here are some tips for starting the week off right (and lowering your stress levels in the process):

• Sleep: Make sure you stick to your usual sleep schedule over the weekend. Sleeping in on Saturday and Sunday can disrupt your usual sleep-wake cycle, and that in turn can raise your cardiovascular risk.
• Support your gut. Oscillations of gut microbes throughout the day are crucial for regulating the secretion of stress hormones. An imbalance in the gut microbiome leads to a hyperactivation of the hypothalamic-pituitary-adrenal (HPA) axis.
• Meditation: Try meditating on Sunday evening, even if it’s just for 5 minutes. Meditation can help reduce stress and support your cardiovascular health.
• Exercise: Get some exercise first thing Monday morning. It will help clear your mind, elevate your mood and reduce your stress levels. Plus, it’s great for your heart.
• Fun: Schedule something enjoyable on Monday, whether it’s time for reading your favorite book, having coffee with a friend before work or walking at lunch with office mates. This will help you look forward to Monday instead of dreading it.
• Natural stress-relieving adaptogenic herbs, like holy basil and ashwagandha, are effective at helping balance your system during stressful periods.

Sources:

New research shows Monday stress is etched into your biology — ScienceDaily

Are anxious Mondays associated with HPA-axis dysregulation? A longitudinal study of older adults in England — Journal of Affective Disorders

The post A biological reason to hate Mondays: It’s a stress amplifier appeared first on Easy Health Options®.

It’s a serious disease that can cause blindness, foot amputation, nerve damage and kidney problems, to name a few of the possible long-term effects.

And even though diabetes doesn’t just appear out of thin air, it can sneak up on you…

Approximately 1 in 3 American adults have prediabetes, a condition where blood sugar levels are higher than normal, but have not crossed the threshold for diabetes yet.

Unfortunately, most people in this stage don’t know the warning signs of prediabetes, that it carries a serious threat for heart damage or that it’s a critical point for stopping the trajectory to type 2 diabetes.

Physicians will advise keeping weight, blood sugar and cholesterol levels healthy through diet and exercise to avoid prediabetes.

But if you still find yourself in prediabetes limbo, you could benefit from extra help — like taking a common vitamin found to slash the risk of progression to full-blown diabetes…

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Vitamin D could save millions from a diabetes diagnosis

Vitamin D, known as the sunshine vitamin, is best known for powerful immune support. Few of us realize it also has important roles in insulin secretion and glucose metabolism. In fact, previous research has associated low blood levels of vitamin D with a higher risk of developing diabetes.

Researchers from Tufts Medical Center conducted a review and meta-analysis (they combined the statistical results) of three clinical trials comparing the impact of vitamin D supplements on diabetes risk for adults with prediabetes.

Over a three-year follow-up period, they saw that supplementing vitamin D was associated with a 15 percent decreased likelihood for developing type 2 diabetes in adults with prediabetes.

Given the fact that more than 374 million adults worldwide have prediabetes, the authors used their findings to deduce that vitamin D supplementation could delay the development of diabetes in more than ten million people.

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Vitamin D helps take down risk factors

If you know a little more about vitamin D’s association with other risk factors that are known to contribute to the development of diabetes, there’s no mystery about why D is the anti-diabetes vitamin…

Take obesity, considered one of the top risk factors. People who are low in vitamin D are also more likely to be obese. But worse, a study from Spain has shown that lacking vitamin D expands your chances of developing diabetes even more than being overweight.

Another study found that people with vitamin D blood levels of 50 ng/mL (considered adequate) were five times less likely to develop diabetes than those with 20ng/mL. The lead author of that study, Dr. Cedric Garland, believes rather than focusing solely on obesity and sugar and fat intake, we should be focusing on vitamin D levels.

Per the good folks at Harvard T. Chan School of Public Health, the RDA for vitamin D provides the daily amount needed to just maintain healthy bones and normal calcium metabolism in healthy people. That means 600 to 800 IU a day likely won’t turn prediabetes around.

The Institute of Medicine increased the upper limit of vitamin D to 4000 IU a day for adults, but other experts recommend a therapeutic daily dose of 5000 IU. Because of a growing body of research that’s finding vitamin D is not a one-size-fits-all vitamin, some have also suggested that dosage be based on BMI.

If you’re unsure what your vitamin D levels are, your doctor can test them. He’d also best know of any conditions or medications that could lower your levels or potential interactions.

Sources:

Vitamin D supplementation may lower diabetes risk for the more than 10 million adults with prediabetes — Eureka Alert

Vitamin D and Risk for Type 2 Diabetes in People With Prediabetes — Annals of Internal Medicine

Time out: should vitamin D dosing be based on patient’s body mass index (BMI): a prospective controlled study — Cambridge University Press

The post The vitamin that could turn prediabetes around appeared first on Easy Health Options®.

But growing research shows a powerful connection: the same choices that protect your heart may also strengthen your bones — and vice versa.

Shared Risk Factors Across Systems

Osteoporosis and cardiovascular disease may seem like two completely different conditions, but they often share the same root causes. Poor diet, a lack of physical activity, smoking, and chronic inflammation can all increase your risk for both. 

Research has shown that people with low bone mineral density are more likely to experience heart-related events. Likewise, signs of atherosclerosis —hardening and narrowing of the arteries — are more common in individuals with weaker bones.

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Lifestyle and Fracture Risk

One large U.S. study looked at how cardiovascular health scores — based on the American Heart Association’s Life’s Essential 8 — related to fracture risk. These eight key health factors include diet, physical activity, sleep, smoking, body weight, cholesterol, blood sugar and blood pressure.

The study found that people with better scores had a significantly lower risk of bone fractures. Compared to individuals with poor cardiovascular health, those with moderate scores had a 22% lower fracture risk, and those with high scores had a 34% lower risk.

This tells us that small steps you take to support your heart — like going for a daily walk or choosing whole foods over processed ones — can also help keep your bones strong.

How Inflammation and Hormones Affect Both Heart and Bone Health

Research has shown that heart disease and osteoporosis often develop together, and inflammation and hormones are two key reasons why.

Chronic inflammation, which plays a major role in heart disease, also contributes to bone loss. When inflammation levels are high, the body produces chemicals that can weaken bone and damage blood vessels at the same time. This means that the same internal stress that leads to clogged arteries can also make bones more fragile.

Hormonal changes, especially in women, add another layer of connection. Estrogen helps protect both the heart and the bones. After menopause, when estrogen levels drop, the risk for both heart disease and osteoporosis accelerates. That’s why, later in life, women often experience both conditions.

The overlap goes deeper. According to research, calcium deposits found in arteries and bone loss seen in osteoporosis may actually be driven by some of the same biological processes. In other words, the breakdown of bone and the buildup of plaque in blood vessels may be two sides of the same coin.

It all serves as a reminder that the body works as an interconnected system. Supporting hormone health and reducing inflammation through healthy habits (and hormone replacement therapy when appropriate) can go a long way in protecting both your heart and your bones.

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Statins and Bone Health

Statins are best known for lowering cholesterol, but they could offer benefits for bones too. Although confounding variables may be at play, several studies suggest that statins can stimulate the growth of bone-forming cells and reduce bone breakdown.

Statins appear to increase levels of a key protein that helps bones grow, while also lowering inflammation and possibly enhancing estrogen activity. These effects have been linked to improved bone mineral density and a reduced risk of fractures, especially in older adults and postmenopausal women.

Having said that, it is possible that these findings are associations rather than causations. People who take statins may be under more regular medical care, might be more attentive to their health in general, and might be more likely to pursue an active lifestyle. The jury is still out on how helpful statins are to bone health. But while more research is needed to fully understand the best use of statins for bone support, these findings suggest that statins certainly do not promote bone loss!   

A Whole-Body Approach to Better Health

When it comes to protecting your health, there’s no need to compartmentalize. Your bones and your heart benefit from the same healthy habits:

• Eat a whole-food, anti-inflammatory diet, rich in fruits, vegetables, whole grains, and healthy fats.
• Get regular physical activity, including weight-bearing and resistance training, which supports both cardiovascular fitness and bone density.
• Don’t smoke, and take steps to manage blood pressure, blood sugar, and cholesterol.
• Talk to your doctor about your risk factors and whether bone density testing is right for you. By the way, this last piece of advice applies to everyone. Although less likely, men with risk factors can get osteopenia and osteoporosis too.

The post The hidden link between heart health and bone strength appeared first on Easy Health Options®.

Being low on magnesium can put you at risk for conditions ranging from low thyroid, asthma and migraines to diabetes, hypertension, and stroke.

But a magnesium deficiency is notoriously hard to detect… until you’ve got one.

Most of our magnesium is stored in our bones, undetectable by routine blood tests. However, it’s estimated that about half of adults in the U.S. are living with a magnesium deficiency.

That means, if you are generally healthy, you could have a magnesium deficiency and not even know it until things get serious.

Now, we can add yet another reason to make sure you have the magnesium your body — and your brain — need.

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More magnesium makes brains younger

Scientists at the College of Health and Medicine at the Australian National University are suggesting that increasing our magnesium intake through a balanced diet can help keep our brains younger and reduce the risk of dementia as we age.

Their study of more than 6,000 cognitively healthy adults aged 40 to 73 showed that those who consumed more than 550mg of magnesium daily had a brain age approximately one year younger than those who consumed only 350mg of magnesium per day (considered the normal intake).

In the younger subjects, this difference was evident by the time they reached age 55.

“Our study shows a 41 percent increase in magnesium intake could lead to less age-related brain shrinkage, which is associated with better cognitive function and lower risk … of dementia in later life,” says lead author Dr. Khawlah Alateeq.

Magnesium counts at every age, especially for women

Female subjects in this study seemed to benefit even more than males from the neuroprotective effects of additional magnesium in their diets.

Considering that women make up about two-thirds of dementia cases, that’s not surprising.

However, it’s clear that we should all be paying attention to the amount of magnesium in our diets.

The researchers say that a higher intake of magnesium in our diets from a younger age may safeguard against neurodegenerative diseases and cognitive decline by the time we reach our 40s.

It really is like an “insurance policy” against dementia.

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LOTS of ways to get more magnesium

There’s nothing wrong with taking a daily magnesium supplement if you’re not sure you’re getting enough.

But foods rich in this mineral are abundantly available. They include:

• Spinach
• Black beans
• Almonds
• Cashews
• Pumpkin seeds
• Avocado
• Bananas
• Broccoli
• Brussels sprouts
• Brown rice
• Low-fat yogurt
• Dried figs
• Dark chocolate

It’s easy to incorporate these magnesium-rich foods into your diet. Add some nuts and bananas to your yogurt. How about brown rice and spinach with your dinner? The possibilities are endless! (Just be sure to rinse your rice first to cut down on heavy metals.)

Also, elevate your snack game with almonds, cashews, dried figs and dark chocolate!

Make sure also to get enough vitamin D3, B6, and E, as well as thiamine and selenium. All of these support your body’s use of magnesium.

Sources:

A higher dose of magnesium each day keeps dementia at bay Science Daily

Dietary magnesium intake is related to larger brain volumes and lower white matter lesions with notable sex differences European Journal of Nutrition

The post The mineral that works like ‘insurance’ against dementia appeared first on Easy Health Options®.

She immediately followed the doctor’s instructions for diet and supplements, but she had one question to which the doctor couldn’t give a satisfying answer:

“Is there anything I could have done to prevent this?”

Right now, the short answer to that is no. AMD has no known cause, and while there are treatments to slow progression, there is no cure.

But scientists are looking to change that. They’re digging deeper into the mechanisms of the disease — and what they’re discovering is fascinating…

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The key could be a molecule that maintains healthy cholesterol

An international team of researchers led by the Washington University School of Medicine in St. Louis (WashU Medicine) used human plasma samples and mouse models of AMD to explore a specific link with cardiovascular disease, both of which worsen with age.

The reason they chose to focus on that connection is that in AMD, doctors can see cholesterol-rich deposits under the retina during an eye exam, according to senior author Dr. Rajendra S. Apte, a professor at WashU Medicine.

While vision might still be normal in the early stages, the deposits increase inflammation and other damaging processes that lead to a gradual loss of central vision.

In recent years, investigators have discovered that a molecule called apolipoprotein M (ApoM) exhibits anti-inflammatory effects and plays a role in maintaining healthy cholesterol metabolism. Levels of ApoM tend to fall with age.

Dr. Apte and co-senior author Dr. Ali Javaheri, also a professor at WashU Medicine, aimed to investigate whether reduced levels of ApoM contribute to the dysfunctional cholesterol metabolism underlying multiple diseases of aging, including AMD and heart disease.

They demonstrated that patients with AMD have lower levels of ApoM in their blood compared to healthy patients. And past research by Javaheri showed patients with various forms of heart failure also had lower levels of ApoM.

This study revealed that ApoM is a key component in the “good cholesterol” pathways that clean up excess “bad” cholesterol — the kind that tends to drive inflammation — and clear it from the body through the liver.

This suggests that when ApoM levels are low, cells in the retina and heart muscle are unable to metabolize cholesterol deposits correctly and have difficulty clearing the accumulating lipids. When they build up, it leads to inflammation and cellular damage.

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Effects of low ApoM could be reversible

To see if they could reverse the harmful effects of low ApoM, the researchers increased ApoM levels in mouse models of macular degeneration. The mice showed evidence of improved retinal health, improved function of light-sensing cells in the retina and reduced accumulation of cholesterol deposits.

In short, the results suggest increasing ApoM in the blood could fix problems in cholesterol processing that lead to cellular damage in the eyes and other organs.

“Our study points to a possible way to address a major unmet clinical need,” Apte says. “Current therapies that reduce the chance of further vision loss are limited to only the most advanced stages of macular degeneration and do not reverse the disease. Our findings suggest that developing treatments that increase ApoM levels could treat or even prevent the disease and therefore preserve people’s vision as they age.”

The findings also could have implications for raising ApoM in patients with heart failure.

You may be wondering if there’s any way we can support our own ApoM levels. There is some evidence that berberine, a naturally occurring compound found in certain plants, may increase ApoM levels to protect the gut-vascular barrier. Sources of berberine include European barberry, goldenseal, goldthread, Oregon grape, phellodendron and tree turmeric.

Berberine is available in supplement form, but be aware that it interacts with several medications. It’s important to check with your doctor first before using it.

Another study in mice showed that niacin increased ApoM gene and protein expression levels. This vitamin is present in foods such as yeast, milk, meat and cereals. Although you can take niacin supplements, you’re likely getting enough of the vitamin from your diet.

If you do take a niacin supplement, don’t take more than what is recommended, or you could end up causing another problem for your eyes — a rare toxic reaction called niacin-induced maculopathy.

Sources:

The molecule that might save your sight—and your heart — ScienceDaily

Apolipoprotein M attenuates age-related macular degeneration phenotypes via sphingosine-1-phosphate signaling and lysosomal lipid catabolism — Nature Communications

Berberine reduces gut-vascular barrier permeability via modulation of ApoM/S1P pathway in a model of polymicrobial sepsis — Life Sciences

Berberine – Uses, Side Effects, and More — WebMD

Niacin regulates apolipoprotein M expression via liver X receptor‑α — Molecular Medicine Report

Niacin — Mayo Clinic

The post A cholesterol cure to save your heart and your vision appeared first on Easy Health Options®.

This means that a person born in 2000 could, on average, live 30 years longer than someone born in 1900, reaching an age of around 80. However, we may have reached an upper limit on human longevity.

In certain countries, including the U.S., the average lifespan has declined slightly in recent years, partly due to our inability to slow the effects of aging, such as frailty, dementia, heart disease and sensory impairments.

Still, scientists continue to explore ways to mitigate the impacts of aging, including high-intensity exercise, hyperbaric oxygen therapy, and supplements such as astaxanthin, pyrroloquinoline quinone and grape seed extract. Each of these methods is believed to activate distinct “longevity switches” in the body, potentially extending lifespan.

Recently, however, researchers may have identified the simplest and most ancient longevity switch of all…

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The impact of caffeine on AMPK

We’ve long known about the potential life-lengthening benefits of coffee, including reduced risk of age-related diseases. However, we haven’t been entirely clear on the mechanism behind it all.

A few years ago, researchers at Queen Mary University of London found that caffeine helps cells live longer by acting on a growth regulator called TOR, or Target of Rapamycin. This biological switch, which tells cells when to grow based on the availability of food and energy, has been controlling energy and stress responses in living things for over 500 million years.

In a recent study, the same research team sought to investigate the effect of caffeine on fission yeast, a single-celled organism closely related to human cells. In doing so, they were surprised to learn that caffeine doesn’t act directly on TOR. Instead, it activates the AMPK system, which acts as a sort of cellular energy monitor and is evolutionarily conserved in both yeast and humans.

“When your cells are low on energy, AMPK kicks in to help them cope,” explains Dr. Charalampos (Babis) Rallis of Queen Mary University of London, the study’s senior author. “And our results show that caffeine helps flip that switch.”

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How caffeine supports our cells

Using the yeast model, the researchers demonstrated that caffeine’s effect on AMPK influences how cells grow, repair their DNA and respond to stress. All of these factors are tied to aging and disease.

“These findings help explain why caffeine might be beneficial for health and longevity,” says Dr. John-Patrick Alao, the postdoctoral research scientist leading this study. “And they open up exciting possibilities for future research into how we might trigger these effects more directly — with diet, lifestyle or new medicines.”

Interestingly, AMPK is also a target of metformin, a common diabetes medication being studied in conjunction with the antifungal agent rapamycin for its potential to extend human lifespan.

Clearly, more research is needed before we can definitively say that caffeine helps us live longer. However, we do know that caffeine has several other health benefits.

For instance, caffeine can help you burn more fat while exercising and fight off Alzheimer’s disease. It protects heart health by lowering LDL cholesterol and reducing the risk of heart failure and atrial fibrillation. It can also lead to reduced dementia risk, prevention of the brain deterioration caused by Parkinson’s disease, lower risk of type 2 diabetes and reduced odds of colon cancer recurrence.

With all these positives, it’s safe to say that you’ll want to keep up your morning coffee habit, or adopt one if you don’t currently indulge.

That said, if you find that the caffeine in coffee makes you too jittery, try a cup of green tea instead, as I do. I find the caffeine in green tea to have a much lighter touch. And the antioxidants in green tea may help ward off cancer, protect your eyesight and balance your blood sugar, all things that can go a long way in helping you live a long, healthy life.

Sources:

Scientists reveal your morning coffee flips an ancient longevity switch — ScienceDaily

Dissecting the cell cycle regulation, DNA damage sensitivity and lifespan effects of caffeine in fission yeast — Microbial Cell

Human Longevity May Have Reached its Upper Limit — Scientific American

What doctors wish patients knew about falling U.S. life expectancy — AMA

The post The cellular longevity switch tripped by caffeine appeared first on Easy Health Options®.

Unfortunately, there is currently no better way to save a damaged or infected tooth than to undergo a root canal. So, it’s best to do everything you can to avoid having one in the first place.

Of course, good dental care is your first line of defense against the kind of tooth infection that leads to a root canal. But who couldn’t use an edge to stay out of the dentist’s chair…

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Exercise, omega-3 and tooth health

Caries, or tooth decay, can cause inflammation at the tip of the tooth’s root, or apex, and its surrounding area. If left untreated, the bacteria can reach the root canal and pass through it to the apex, causing an infection known as apical periodontitis. A form of gum disease, this condition can lead not only to tooth loss but also to bone loss.

“It’s a condition that patients may not even know they have because of its chronic nature, but which can evolve and lead to bone destruction and tooth mobility,” says Rogério de Castilho of the Araçatuba School of Dentistry at São Paulo State University (FOA-UNESP) in Brazil. “In addition, in specific situations, such as a drop in immunity, it can become acute, so the patient starts to feel pain, pus forms at the site, the face can become swollen.”

There’s also a two-way relationship between specific health conditions that can worsen apical periodontitis, including diabetes, metabolic syndrome, arteriosclerosis and kidney disease. At the same time, infection in the apex can exacerbate these diseases.

Castilho supervised a study in which researchers at FOA-UNESP induced apical periodontitis in 30 rats and divided them into three groups. The first group was left alone. The second and third groups underwent a 30-day swimming regimen. The third group also received dietary supplementation of omega-3, a polyunsaturated fatty acid that has proven therapeutic effects on chronic inflammation and associated diseases.

“In rats, physical exercise alone brought about a systemic improvement, regulating the local immune response,” says Ana Paula Fernandes Ribeiro, the first author of the study, carried out during her doctorate at FOA-UNESP. “In addition, when combined with supplementation, it further reduced the destructive condition caused by endodontic pathology.”

Benefits to bone and tissue

The study is the first to show that a combination of moderate physical exercise and omega-3 supplementation significantly reduces the inflammation caused by apical periodontitis. The combination of these activities:

• Limited bacterial progression.
• Reduced bone tissue loss.
• Regulated the release of pro-inflammatory cytokines.
• And stimulated the activity of fibroblasts, the cells that create and maintain tissue.

The authors note that the study provides new evidence of the benefits of exercise and omega-3 fatty acids for the immune system and oral health. Next, they would need to conduct a large clinical study to see if the same would be true in humans.

However, this study reinforced the findings of previous research that indicate omega-3 fatty acids could help fight off periodontitis and protect against the tooth pulp infection and loss that lead to root canal.

Keeping your teeth healthy could be the nudge you need to exercise regularly and add healthy omega-3s to your diet. Make sure you take a high-quality fish or krill oil supplement, or if you’re a fan of fish, eat at least two servings weekly.

Here are a few fish that are rich in omega-3:

• Mackerel
• Salmon
• Herring
• Oysters
• Sardines

Sources:

A combination of exercise and omega-3 reduces the severity of tooth root infections — Agência FAPESP

Physical exercise alone or combined with omega-3 modulates apical periodontitis induced in rats — Scientific Reports

What is a Root Canal? — American Association of Endodontists

The post The edge that could keep you out of the dentist’s chair appeared first on Easy Health Options®.

That’s why a lot of women choose to take hormone replacement therapy (HRT) to counteract some of those symptoms. But HRT isn’t for everyone, and may elevate risk of breast, endometrial and ovarian cancers, as well as heart attack, stroke and pulmonary embolism.

But what if I told you there was an all-natural way to practically eliminate one of the most bothersome symptoms of menopause?

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The vegan diet and hot flashes

About 80 percent of menopausal women suffer from vasomotor symptoms commonly known as hot flashes. They can occur during the day or night (or worse, both), interfering with sleep and quality of life.

Back in 2022, researchers looking for ways to counteract these uncomfortable symptoms conducted the Women’s Study for the Alleviation of Vasomotor Symptoms (WAVS). The study involved 84 women who reported having two or more moderate-to-severe hot flashes daily.

The women were randomly assigned to one of two groups: an intervention group asked to follow a low-fat vegan diet that included a half cup of cooked soybeans a day, or a control group that continued their usual diets for 12 weeks.

The results were remarkable. The vegan diet…

• Decreased overall hot flashes by 95 percent
• Eliminated severe hot flashes
• Led to a 96 percent decrease in moderate-to-severe hot flashes
• Reduced daytime and nighttime hot flashes by 96 and 94 percent, respectively

As if that weren’t enough, participants following the vegan diet lost 6.4 pounds on average.

“We do not fully understand yet why this combination works but it seems that these three elements are key — avoiding animal products, reducing fat, and adding a serving of soybeans,” explains lead researcher Neal Barnard, MD, president of the Physicians Committee and adjunct professor at the George Washington University School of Medicine.

“Our results mirror the diets of places in the world, like pre-Westernized Japan and modern-day Yucatán Peninsula, where a low-fat, plant-based diet including soybeans is more prevalent and where postmenopausal women experience fewer symptoms.”

But to get more insight into how a vegan diet affected these changes, the researchers conducted a secondary analysis of the WAVS data — and have recently released their findings…

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Changes to the gut microbiome made all the difference

The researchers discovered something unusual in the gut microbiomes of the women that could account for the relief provided by the vegan diet…

When comparing stool samples from a subset of 11 participants at the start of the study and again after 12 weeks on a vegan diet, they found changes in the amount of several types of gut bacteria.

They saw an association between decreased severe daytime hot flashes and a reduction in the abundance of Porphyromonas and Prevotella corporis bacteria. Prevotella corporis has also been found in the gut of rheumatoid arthritis patients and appears to have pro-inflammatory properties.

In addition, a decrease in Clostridium asparagiforme was linked with a reduction in total severe and severe nighttime hot flashes. This bacteria has been shown to produce trimethylamine-N-oxide, a compound associated with an increased risk of cardiovascular disease and type 2 diabetes.

Therefore, a reduced abundance of Clostridium asparagiforme could partly explain the benefits of a plant-based diet for cardiovascular health. It could also provide a possible link between hot flashes and cardiovascular disease. For women, the risk of heart disease begins to climb during menopause and skyrockets in the years following.

The researchers also found changes in the levels of other bacteria could play a role in alleviating hot flashes by stabilizing estrogen levels, reducing inflammation and increasing satiety.

“Women who want to fight hot flashes should feed the bacteria in their gut a vegan diet rich in fruits, vegetables, grains and beans, which also leads to weight loss and protects against heart disease and type 2 diabetes,” says study co-author Dr. Hana Kahleova, director of clinical research at the Physicians Committee for Responsible Medicine.

Sources:

1. Vegan diet fosters changes in gut microbiome that reduce hot flashes by 95%, finds new study — EurekAlert!

2. A dietary intervention for postmenopausal hot flashes: A potential role of gut microbiome. An exploratory analysis — Complementary Therapies in Medicine

3. Hot flashes — Mayo Clinic

The post 3 ingredients that stop hot flashes, start weight loss in menopause appeared first on Easy Health Options®.

While the exact reason why has been a mystery, there are a couple of theories…

Women tend to live longer than men, and the most significant risk factor for Alzheimer’s is age. It skyrockets in those aged 75 and older.

Also, women are twice as likely as men to have an autoimmune disease. Since some experts say beta-amyloid is a normally occurring molecule that is part of the brain’s immune system, an overactive immune system seems a logical connection.

Changes in hormones have also linked Alzheimer’s to women, not only the loss of estrogen, but higher levels of cortisol in midlife as well.

Fortunately, recent research may not only explain why women are at higher risk of the disease — but also lead to the development of a simple blood test that could detect Alzheimer’s in the early stages…

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The roles of acetyl-L-carnitine and free carnitine

An international team of researchers measured blood levels of two amino acids essential for healthy brain function — acetyl-L-carnitine and free carnitine — in two separate groups of men and women in Brazil and California. Some were diagnosed with varying degrees of cognitive impairment, while others were cognitively healthy.

The researchers discovered that blood levels of acetyl-L-carnitine were lower in both women and men with mild cognitive impairment and Alzheimer’s disease. In addition, levels of free carnitine, a byproduct of acetyl-L-carnitine in reactions essential to brain function, showed a steady decline in women in amounts related to the severity of their cognitive decline.

Tests also indicated that levels of the two molecules aligned in direct proportion to increased levels of amyloid beta and tangled tau protein, both of which are long considered markers of Alzheimer’s severity.

The research team’s accuracy in diagnosing Alzheimer’s severity rose from more than 80% when using either the two blood molecules or amyloid beta and tangled tau protein levels from cerebrospinal fluid, to 93% when using both.

The fact that declines in men were only evident in acetyl-L-carnitine reveals a disease-specific difference between the sexes and could explain the higher risk of Alzheimer’s in women.

“Our findings offer the strongest evidence to date that decreased blood levels of acetyl-L-carnitine and free carnitine could act as blood biomarkers for identifying those who have Alzheimer’s disease, and potentially those who are at greater risk of developing early dementia,” says study lead investigator Dr. Betty Bigio, a professor at NYU Grossman School of Medicine.

“Because declines in acetyl-L-carnitine and free carnitine tracked closely with the severity of Alzheimer’s disease, the molecular pathways involved in their production offer other possible therapeutic targets for getting at the root cause of the disease and potentially intervening before permanent brain damage occurs,” said senior study investigator Carla Nasca, PhD. Nasca is an assistant professor in the Departments of Psychiatry and Neuroscience at NYU Grossman School of Medicine.

Dr. Nasca suggests that further research is needed to clarify the role of acetyl-L-carnitine in brain chemistry. If further studies confirm the latest findings, she adds, they could be used to develop a simple blood test for dementia and to track the progression of Alzheimer’s.

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Getting enough acetyl-L-carnitine

Previous studies have found that acetyl-L-carnitine can help improve Alzheimer’s symptoms by enhancing cognitive function and mitigating cognitive impairment.

One study found memory and cognitive scores in Alzheimer’s patients given 2,250 to 3,000 mg daily of acetyl-L-carnitine improved 2.8 times better than placebo-treated subjects over 12 weeks.

Carnitine is produced naturally by the body through synthesis by the liver and kidneys. However, levels of carnitine in the body do decline with age, with one study finding levels were about 20% lower in aged rats.

So if you’re older, it may be a good idea to consume foods rich in carnitine. These include animal products such as red meat, poultry, fish and dairy products. Fruits, vegetables and grains contain negligible amounts of the amino acid.

You could also try acetyl-L-carnitine supplements. Just be careful if you’re taking blood thinners or drugs for lowering blood sugar — you’ll want to talk to your doctor first before adding acetyl-L-carnitine to the mix.

Sources:

Study advances possible blood test for early-stage Alzheimer’s disease — EurekAlert!

Sex differences in mitochondrial free-carnitine levels in subjects at-risk and with Alzheimer’s disease in two independent study cohorts — Molecular Psychiatry

Acetyl-L-Carnitine in Dementia and Other Cognitive Disorders: A Critical Update — Nutrients

Why are women more likely to develop Alzheimer’s disease? — Harvard Health Publishing

Acetyl-L-carnitine supplementation restores decreased tissue carnitine levels and impaired lipid metabolism in aged rats — Journal of Lipid Research

Carnitine — National Institutes of Health

The post Two molecules explain Alzheimer’s ‘mystery’ and enable early detection appeared first on Easy Health Options®.

After all, who would voluntarily dunk themselves in cold water? A man named Wim Hof, for one. He’s known as the Iceman, and his method of cold exposure may sound crazy, but crazy like a fox.

Surprisingly, research has consistently shown that cold water immersion offers numerous health benefits, including reduced body fat, which can improve blood glucose levels. It also appears to mimic the effects of calorie restriction, which is associated with increased lifespan.

If that last part got your attention, you’ll be interested in what researchers learned when they took a dive into how cold water impacts aging at the cellular level.

Here’s what you need to know (plus, a way to grab these benefits that won’t make your teeth chatter)…

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Boosting cellular resilience and autophagic function

Research conducted at the University of Ottawa, which has expertise in cold-related studies, explored the effects of cold water acclimation on autophagic and apoptotic responses.

In simple terms, they sought to learn more about the effects of frigid temperature on cell health, cell recycling and programmed cell death, to support youthful health and vitality.

The researchers collected blood from participants who took a one-hour cold water plunge (57.2°F) each day for seven days.

The results?

“Our findings indicate that repeated cold exposure significantly improves autophagic function, a critical cellular protective mechanism,” says Professor Glen Kenny. “This enhancement allows cells to better manage stress and could have important implications for health and longevity.”

Additionally, those cold plunges led to a decrease in blood markers related to cellular damage.

Overall, the scientists suggest that cold exposure, which enhances autophagic activity, may not only prolong cellular longevity but also prevent the onset of various diseases and potentially slow down aging at a cellular level.

They even went so far as to call it “a tune-up for your body’s microscopic machinery!”

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Looking beyond cold to power longevity

As great as that sounds, there are probably not many of us willing to jump into a cold bath regularly in the hopes of grabbing those benefits.

What else can you do that’s also backed by research to promote extra years of healthier aging that could increase your odds of a longer lifespan?

Get active. In one study, people who were the most active had an incredible 73% lower risk of death than their least active counterparts. Walking counts, especially if you do it at a fast pace.

Supplement smart:

• Vitamin D was found to reduce biological aging by three years during a four-year study.
•  Pyrroloquinoline quinone (PQQ) tops the list of 10 micronutrients that turn the key on age reversal by supporting the cellular powerhouses known as mitochondria. One of those is also an old favorite…
• Omega-3 fatty acids, which support the structure and function of mitochondrial membranes.

Lastly, cultivate relationships. Many studies have shown that loneliness is associated with factors that can shorten lives, while having friendships cultivates good health and promotes longevity.

Cold water immersion may seem intimidating, but its potential benefits for cellular health and longevity are noteworthy. However, it’s certainly not for everyone.

Fortunately, a healthy, long life can also be achieved through regular exercise, a good diet, smart supplementation and nurturing relationships. By embracing this holistic approach, you can not only promote healthy aging but also tap into the potential for longevity.

Sources:

Cold plunges actually change your cells — ScienceDaily

The post Cold water immersion: Adapting your cells to live longer appeared first on Easy Health Options®.

Sounds too good to be true, right? Well, think again.

Chances are that if you have heard of this supplement, it was in the context of high-performance athletes.

But it’s actually a vital nutrient for anyone who wants to support everyday wellness and healthy aging. Does that sound like you?

Then, read on for the skinny on creatine and why you need more of it.

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Creatine: What is it

Creatine is a nutrient, a compound to be exact, produced by three amino acids. Its function is to increase the amount of adenosine triphosphate (ATP) in our cells — the body’s main source of energy.

Creatine can be found in seafood and red meat. It’s also available in flavored or unflavored powders, both in health stores and online.

In the gym, supplementing creatine translates to enhanced energy production, which means more energy to build muscle faster and to recover faster.

In general, ATP stores energy and delivers it to various parts of the body as it is needed to support everything from digestion to movement to thinking.

Once seen as a supplement for “gym rats” only, experts now say creatine plays a vital role in cellular energy, cognitive function, and healthy aging — and we should all take advantage of it.

What it does

If you’re dealing with physical fatigue that prevents you from staying active or finding that you’re just not as sharp as you once were, supplementing with creatine could help.

Dr. Richard Kreider, professor and director of the Exercise and Sport Nutrition Lab at Texas A&M University, has spent over 30 years investigating the effects of creatine.

He says, “When the body is stressed, like in exercise or under metabolic conditions like some diseases, creatine phosphate is needed to maintain energy in the cell, and therefore has a lot of protective and health benefits, in addition to the exercise performance effects that have been seen.”

But studies have found that most people aren’t getting enough. Women see a drop in their body’s creatine stores during hormonally driven periods such as menopause, menstruation or pregnancy.

And, as we age, we produce and store less. Production of ATP also slows down.

This slowing down of production has a particularly noticeable effect on muscle mass, strength and cognitive function.

In fact, just a couple of years ago, a critical analysis of nutritional supplements for healthy aging suggested four nutrients that could help prevent sarcopenia — the loss of muscle mass that occurs with age — and one of them was creatine.

Low dietary creatine intake is also associated with higher body fat. So why are we just hearing about its benefits outside of the gym?

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Unfounded concerns over safety and bloating

For years, there have been misgivings about its safety and the potential for uncomfortable bloating. But Dr. Kreider says a vast body of research shows it’s safe, effective and well-tolerated by most people.

When he and his colleagues analyzed 685 clinical trials on supplementing with creatine to assess its safety, their analysis revealed no significant differences in the rate of side effects between subjects taking creatine and those taking a placebo.

“There’s absolutely no data supporting any negative side effect anecdotally reported about creatine on the internet and in the media,” Dr. Kreider says. “Creatine is safe, and it’s important for everybody, not just bodybuilders and athletes.”

The problem is, most of us fall short in getting enough from our diets.

“You only get about a gram of creatine per pound of red meat or fish, like salmon, so it’s expensive and takes a lot of calories to get a gram,” Kreider said.

There’s no clearly defined Recommended Dietary Allowance for creatine, but the standard recommendation is 3 to 5 grams per day. Dr. Kreider suggests consuming 5 to 10 grams per day will maintain healthy stores and provide enough to support the brain.

When I try a new supplement, I usually start out with the lowest recommended amount and work my way up to see how well I tolerate it. I think that’s fair advice for just about anyone.

If you have kidney problems, it’s a good idea to discuss supplementing with your doctor first.

Sources:

This overlooked supplement could help you think sharper and age better — Science Daily

Safety of creatine supplementation: analysis of the prevalence of reported side effects in clinical trials and adverse event reports — Journal of the International Society of Sports Nutrition

Effects of creatine supplementation on cognitive function of healthy individuals: A systematic review of randomized controlled trials — Experimental Gerontology

Creatine shows potential to boost cognition in Alzheimer’s patients — University of Kansas Medical Center

Creatine monohydrate pilot in Alzheimer’s: Feasibility, brain creatine, and cognition — Alzheimer’s Association

The post The ‘gym supplement’ for healthy aging and everyday wellness appeared first on Easy Health Options®.

That describes what happens when the outer layer of our skin becomes thinner, paler and less stretchy — contributing to the signs of aging we see in the mirror.

It’s also why our skin may tear or bruise much more easily and take longer to heal.

The good news is, however, that a simple vitamin, which is appearing more frequently in anti-aging skincare, may actually restore skin function by reactivating genes essential for skin cell renewal…

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The skin-boosting properties of vitamin C

As we get older, our production of skin cells begins to decline. That’s the first step leading to thinner, more fragile skin. Our bodies also produce less collagen, the protein that gives our skin structure and elasticity.

Researchers in Japan decided to test vitamin C, known for its antioxidant properties and commonly used in skincare products, on laboratory-grown models that mimic real human skin to see how the nutrient impacts skin regeneration. The surface of the skin cell model was exposed to air and nourished from underneath by a liquid nutrient medium, replicating the way human skin receives nourishment from underlying blood vessels while remaining exposed to the external environment.

Using this model, the researchers applied vitamin C at concentrations comparable to those typically transported into the epidermis by the bloodstream, and some amazing things began to happen…

Within seven days, the vitamin C-treated skin model exhibited a thicker layer of epidermal cells, without significantly affecting the stratum corneum, the outermost layer of the skin, which is composed of dead cells. By day 14, the inner epidermal layer had become even thicker, while the outer stratum corneum layer had become thinner.

This suggests something remarkable: Vitamin C promotes the formation and division of keratinocytes, the cells that make up 90 percent of the epidermis.

In other words, vitamin C appears to affect skin cell growth on a genetic level by reactivating genes associated with cell proliferation. It does so by promoting DNA demethylation, a process that spurs gene expression and helps cells grow, multiply and differentiate.

“VC seems to influence the structure and function of epidermis, especially by controlling the growth of epidermal cells,” says Dr. Akihito Ishigami of the Tokyo Metropolitan Institute for Geriatrics and Gerontology (TMIG), Japan.

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Using vitamin C in skincare

According to the findings, vitamin C promotes skin renewal by triggering genetic pathways involved in growth and repair. That means vitamin C may be especially helpful for older adults or those with damaged or thinning skin by boosting the skin’s natural ability to regenerate and strengthen itself.

“We found that VC helps thicken the skin by encouraging keratinocyte proliferation through DNA demethylation, making it a promising treatment for thinning skin, especially in older adults,” Ishigami says.

Of course, this research needs to be confirmed by human studies. Until then, however, it couldn’t hurt to add a vitamin C cream or serum to your skincare regimen and make sure you’re getting plenty in your diet.

One thing to remember is that the vitamin C in creams and serum is prone to breaking down when exposed to light, heat or oxygen. That means as soon as you open your vitamin C skincare product, the clock starts ticking on its longevity.

To help extend your vitamin C’s shelf life, make sure the cream or serum is stored in an opaque bottle or jar and seal it tightly after each use. Never store it in the bathroom, where light and humidity can compromise its effectiveness. Instead, store it in the refrigerator, which will help delay the oxidation process. And once the serum turns yellow or brown, it means it’s oxidized, so discard it.

Sources:

Vitamin C flips your skin’s “youth genes,” reversing age-related thinning — ScienceDaily

Vitamin C Promotes Epidermal Proliferation by Promoting DNA Demethylation of Proliferation-Related Genes in Human Epidermal Equivalents — Journal of Investigative Dermatology

Wrinkles — Cleveland Clinic

How to Store Vitamin C Serum — wikiHow

The post The vitamin scientists call a ‘game changer’ for aging skin appeared first on Easy Health Options®.

Flavonoids help prevent diseases like diabetes and heart disease.

So you may think that it’s a good idea to eat as much flavonoid-rich food as you can.

Well, you wouldn’t exactly be wrong there, but ….

“Eat the rainbow” was coined for a good reason: to reap the incredible disease-fighting benefits of flavonoids and reduce your risk of early death, research suggests it’s key to get them from many different sources.

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Diversity matters to live long and without chronic disease

An international team of researchers analyzed data on 124,805 adults in the UK Biobank aged 40 or over, looking for links between self-reported diet and health.

They found a correlation between the amount of flavonoids in the diet and conditions such as type 2 diabetes, cardiovascular disease (CVD), cancer and neurological disease.

According to ECU Research Fellow, first author and co-lead of the study Dr Benjamin Parmenter, “Flavonoid intakes of around 500 mg a day was associated with a 16% lower risk of all-cause mortality, as well as a ~10% lower risk of CVD, type 2 diabetes, and respiratory disease. That’s roughly the amount of flavonoids that you would consume in two cups of tea.”

He added, however, that those who consumed the widest diversity of flavonoids from a variety of foods, even when consuming the same total amount, had an even lower risk of these diseases.

So why does diversity matter?

According to study co-lead Professor Aedín Cassidy from the Co-Centre for Sustainable Food Systems and Institute for Global Food Security at Queen’s University Belfast, “… different flavonoids work in different ways, some improve blood pressure, others help with cholesterol levels and decrease inflammation. This study is significant as the results indicate that consuming a higher quantity and wider diversity has the potential to lead to a greater reduction in ill health than just a single source.”

“The results provide a clear public health message, suggesting that simple and achievable dietary swaps, such as drinking more tea and eating more berries and apples for example, can help increase the variety and intake of flavonoid-rich foods, and potentially improve health in the long-term,” she added.

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How to improve your flavonoid game

There are over 6,000 known flavonoids found in a variety of foods, and they can appear together in some foods, including:

Flavonols like quercetin, found in apples and pears, lower stroke risk and reduce inflammation. Quercetin also slashes dementia risk, along with kaempferol and others.

Isoflavones are a type of phytoestrogen.

Flavanones lower cholesterol and fat in the blood..

Anthocyanins found in purple, red, and blue produce are antioxidants that support brain, heart and immune functions.

Chalcones have antioxidant properties, protecting the body from cancer and age-related illnesses.

Catechins are popularly found in green tea, and play a role in skin and gut health, among other benefits.

Clearly, you want to be eating plenty of flavonoids.

But here’s the thing … these different classes of flavonoids aren’t all found in one place.

They’re spread among a wide range of foods, including:

• Blueberries
• Peaches
• Red grapes
• Cranberries
• Apples
• Kale
• Onions
• Broccoli
• Black and green tea
• Soy beans
• Cherries

Oh, and they’re also found in red wine and dark chocolate!

A challenge for you

The people in this study who were consuming the most flavonoids were snacking on 19 different types of flavonoids a day!

Can you come close to that? Want to try?

The way to do it, or to even come close, is to eat a wide range of flavonoid-rich foods. Some were mentioned above. Others include:

• lettuce
• bananas
• raspberries
• blackberries
• strawberries
• citrus fruits
• tomatoes
• pears

Nutritionists advise you to “eat the rainbow,” meaning you should consume fruits and vegetables of many colors in order to get your share of flavonoids.

And with summer upon us, that shouldn’t be too hard!

Sources:

Having a mix of tea, berries, apples, and grapes every day may lower your risk of chronic disease and early death — Scimex

A Mix of These Specific Foods Could Help You Avoid Chronic Disease — Science Alert

High diversity of dietary flavonoid intake is associated with a lower risk of all-cause mortality and major chronic diseases — Nature Food

Top foods high in flavonoids — Web MD

The post Diversify your flavonoids to ditch disease and live longer appeared first on Easy Health Options®.

It’s a trend, they say, that cannot be explained by genetics alone.

So, what’s to blame?

Could it be due to higher rates of obesity? Maybe it’s lack of exercise?

Or is it something more sinister, such as the chemicals our government allows manufacturers to add to products that come into contact with the food we eat?

Recent research points to the latter…

Potential to cause breast cancer and beyond

As far back as 2007, an organization called Silent Spring published a list of 216 chemicals that can cause breast cancer in rodents.

Of course, if there’s anything the U.S. Food and Drug Administration doesn’t do when it comes to protecting the public, it’s to act quickly.

So, researchers, writing in the journal Frontiers in Toxicology, set out to compare the Silent Springs list with another database known as Food Contact Chemicals Monitored in Humans, or FCChumon, which is a list of food contact chemicals detected in human breast milk, blood, urine, and tissues.

They found that nearly 200 chemicals connected to breast cancer are used in the making of food packaging and plastic tableware, and dozens of those carcinogens can migrate into the human body.

Among them were 642 endocrine-disrupting chemicals that may stimulate estrogen or progesterone production, another known risk factor for breast cancer.

While the study focused on breast cancer-causing toxins, the team found the dangers of the products we use every day to store and eat our food span the cancer universe.

“There is strong evidence that 76 known or potential breast carcinogens from food contact materials recently purchased all over the world can be found in people,” said study coauthor Jane Muncke.

After analyzing different forms of food packaging and tableware, researchers discovered chemicals including:

• Benzene – A known carcinogen linked to breast cancer development
• 4,4’-Methylenebis-(2-Chloroaniline) – Classified as a probable carcinogen that may cause bladder cancer
• 2,4-Toluenediamine – A toxin known to cause breast and other types of cancers in animal studies
•  3,3′-Dimethylbenzidine and o-Toluidine – Dyes used to color plastic and paper, which, according to the EPA, have the potential to metabolize to carcinogenic amines both in and on the human body.
• Forever chemicals – Bisphenols, phthalates or perfluoroalkyl and polyfluoroalkyl substances, known as PFAS, are linked to high cholesterol, cancer and various chronic diseases.

And while researchers determined that most exposure to carcinogens comes from plastics used in food packaging, a whopping 89 suspected carcinogens were found in paper and cardboard containers.

“Paper has additives such as emulsifiers and adhesives, say if papers are glued together, or there’s a plastic layer glued to the paper,” Muncke points out.

Protect yourself from lurking carcinogens

All of those plastic storage bags, containers, and dinnerware, as well as paper plates, that we use because they are so convenient, present a real and present danger to our health.

So, what can you do to combat this danger?

First, banish the plastic from your kitchen. This includes discarding plastic containers and opting for glass containers to store your food.

Also, be sure always to use a glass dish when putting food in the microwave instead of plastic.

Next, replace any plastic water bottles with glass or stainless steel.

For added protection, toss your non-stick pans, which are coated in cancer-causing forever chemicals.

Most importantly, support your body’s natural detoxification processes, which involve your colon and liver.

The liver, of course, is the body’s primary detox organ. But considering this onslaught of toxins, it’s often overworked, especially if you are overweight. Read here about 8+ ways to relieve your liver’s detox load.

Next, check your transit time. That’s the amount of time your colon processes waste to leave the body. If it takes longer than 24 hours, you’re allowing chemicals to sit in your colon up against the intestinal endothelium, where they can leach into your bloodstream.

Sources:

Chemicals linked to breast cancer leach into our foods, study finds — CNN

Risk Management for Benzidine Dyes — EPA

The post The chemical exposure behind early onset breast cancer appeared first on Easy Health Options®.

Yet half of those people don’t even know they have it.

COPD isn’t necessarily a death sentence. It all depends on how early it’s diagnosed and how quickly lung inflammation progresses.

And now, once again, a simple vitamin may be the answer.

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COPD: a quick overview

COPD is an umbrella term for a group of incurable inflammatory lung disorders, including chronic bronchitis and emphysema. It usually comes from long-term exposure to cigarette smoke and air pollution.

As lung inflammation increases, so does air flow in and out of the lungs. Your tissues are absorbing less and less oxygen. Aside from shortness of breath, people with COPD have a higher risk of developing heart disease, stroke, and diabetes.

People with COPD have an increased risk of respiratory infections and pneumonia. Influenza in particular can lead to serious lung infection and, at worst, death.

The potential of vitamin B3

Discovering the potential of vitamin B3 to combat lung inflammation is a ray of hope for COPD patients. According to a study from the University of Copenhagen, vitamin B3 could improve the quality of life for people with COPD.

“In the study, we show that nicotinamide riboside, also known as vitamin B3, can reduce lung inflammation in COPD patients,” says Associate Professor Morten Scheibye-Knudsen, the study’s co-author.

“This is significant, because inflammation can lead to reduced lung function in these patients,” he says.

In this study, 40 patients with COPD received either a placebo or vitamin B3 in doses of 2 grams per day. Those taking vitamin B3 showed a significant decrease in interleukin 8 (IL-8), a marker indicating the presence of inflammation.

After just six weeks, the researchers observed a 53 percent drop in IL8 in those taking B3. This increased to 63 percent after another twelve weeks!

In other words, the group treated with vitamin B3 experienced reduced lung inflammation during the study.

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Getting more B3 in your diet

Prof. Scheibye-Knudsen cautions that larger studies are needed.

“Only through thorough research will we be able to offer the best and most effective treatment to people suffering from this difficult disease,” he says.

But there’s no harm in adding more B3 to your life now…

In fact, the researchers also found that COPD patients have lower levels of NAD, a coenzyme, in the blood, which is associated with accelerated ageing based on DNA methylation levels. Treatment with vitamin B3 resulted in higher NAD levels and showed signs of delaying cellular aging!

To most of us, vitamin B3 is known as niacin. Your body needs it for proper metabolism, nervous system function and antioxidant protection.

What’s more, it’s an essential nutrient. Your body can’t produce it, so you need to get it from your diet.

Fortunately, that’s not hard. There are plenty of niacin-rich foods, including:

• Liver
• Chicken
• Tuna
• Turkey
• Salmon
• Peanuts
• Avocado
• Brown rice
• Whole wheat
• Mushrooms
• Green peas

Cereals and juices are also fortified with niacin. Make sure to include these foods in your diet regularly.

Sources:

New study shows promising results for COPD treatment — Eureka Alert

Effect of nicotinamide riboside on airway inflammation in COPD a randomized placebo controlled trial — Nature Aging

Chronic obstructive pulmonary disease (COPD) — Cleveland Clinic16 foods that are high in niacin (B3) — Healthline

The post The vitamin treatment for COPD that delays cellular aging appeared first on Easy Health Options®.

From food to books and everything in between, you can learn a lot about your family, friends and even yourself by their answers.

It’s why just the other day, I asked myself that same question when it came to supplements. I take several, so if I could only choose one, the answer, for me, is really quite simple…

If I were stuck on a desert island, the supplement I could not do without would be an omega-3. Preferably krill oil (fish oil gives me fish burps), but I digress…

If you’re down to just one choice, you want the nutrient science has consistently demonstrated can fight all inflammatory and metabolic disorders.

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#1 – Inflammation

Doctors now consider inflammation to be “the root of disease.” The good news is that we already have omega-3s to yank out that root and preserve our health.

That’s because studies have shown that the unsaturated fatty acids in these omegas act to directly resolve inflammation. In addition to this direct action, they provide the additional bonus of competing with omega-6s (the kind found in refined oils) in your diet. This action inhibits the synthesis of pro-inflammatory prostaglandins and shifts the balance toward a more anti-inflammatory state.

#2 – Obesity

Obesity, while usually seen through the lens of weight alone, is actually an inflammatory condition that kicks off a multitude of other dangerous diseases. But omega-3s can once again come to the rescue with their potent anti- inflammatory and adipogenic effects that occur right inside the fat tissue itself.

It’s no wonder, then, that studies have found that even mice fed an obesogenic (obesity-inducing) diet benefit from reduced fat mass accumulation when taking omega-3s. To top it off, researchers also discovered that a higher proportion of omega-3s in red blood cell (RBC) membranes has been associated with a lowered fat mass index.

#3 – Heart Health

As with their effects on obesity, larger amounts of omega-3s in RBC membranes support heart health by reducing inflammatory markers and lowering triglycerides.

Numerous prospective and meta-analytic studies have concluded that supplementing with omega-3s in your daily diet substantially reduces the risk of cardiovascular disease by 15 to 25%.

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#4 – Blood Sugar

In addition to their anti-inflammatory properties, omega-3’s also have potent abilities in the fight against metabolic dysfunction, including blood sugar issues. In fact, research has revealed that these fatty acids improve insulin sensitivity by increasing adiponectin secretion, enhance glucose uptake through an increase in GLUT-4 content, and support pancreatic β-cell function, enhancing their insulin secretion capacities.

#5 – Liver Health

Fatty liver disease may also be no match for omega-3s. These fatty acids work to guard liver health by:

• Inhibiting the NF-κB pathway, leading to decreased transcription of pro-inflammatory cytokines and reduced liver inflammation
• Reducing neutrophil infiltration and enhancing the clearance of apoptotic cells to resolve liver inflammation
• Blocking NLRP3 inflammasome activation and the inflammation that comes with it and preventing the progression from simple steatosis (fat buildup) to steatohepatitis (with associated inflammation and liver damage)
• Inducing mitophagy of damaged liver mitochondria and reducing oxidative stress

A comprehensive cohort study even found that taking omega-3s regularly resulted in a 28% reduced risk of liver inflammatory disease, including alcoholic or non-alcoholic liver disease and liver failure.

#6 – Kidney Health

Chronic kidney disease (CKD) is another downstream effect of obesity-mediated inflammation. Luckily, supplementation with omega-3s has been shown to increase the production of pro-resolving mediators, like RvE1 and RvD5, that result in an improvement in inflammatory markers and renal function.

Omega-3s also help improve renal hemodynamics and significantly reduce proteinuria in patients with CKD.

#7 – Autoimmunity

• Inflammatory bowel diseases – Omega-3s have been shown to reduce colitis-associated disease severity, colonic mRNA expression of inflammatory cytokines and macrophage infiltration of the tissue.
• Psoriasis – In psoriatic skin, omega-3s work to reduce inflammatory cell infiltration and skin thickening, with improvements in redness, scaling and itching.
• Rheumatic diseases – Omega-3s also provide powerful joint support by inhibiting IL-1β, one of the most active cytokines that leads to cartilage destruction and loss of collagen.
• Multiple sclerosis – Because omega-3s can cross the blood–brain barrier, they have been shown to directly impair neuroinflammatory processes. Research also shows they may decrease the activity and production of matrix metalloproteinase-9, which is implicated in blood–brain barrier breakdown in MS, while upregulating the expression of brain-derived neurotrophic factor (BDNF) to enhance neuronal survival and synaptic plasticity.

Research has even shown that supplementation with omega-3 fatty acids alone can reduce autoimmune disease rate by 15%.

Putting omega-3s to work for your health

Clearly, whether you end up marooned on a desert island or just want to stay healthy and keep inflammatory and metabolic disease at bay, omega-3s are a must-have in your diet.

So, what’s the best way to ensure you’re getting enough?

One solution is to adopt the Mediterranean diet. Not only is this diet packed with polyphenol-rich fruits, vegetables, and olive oil, but it is also rich in seafood, providing you with those essential omega-3s.

However, if you’re not able to reach the recommended two to three servings of fatty fish weekly, go easy on yourself and reach for a safe, sustainably sourced omega-3 supplement.

Sources:

Beneficial Effects of Omega-3 Fatty Acids on Obesity and Related Metabolic and Chronic Inflammatory Diseases — MDPI

How do Omega-3 and Vitamin D Play a Role in Autoimmune Disease? — Omegaquant

The post The ONE nutrient to fight inflammatory and metabolic disorders appeared first on Easy Health Options®.

As soon as an additive is linked to health issues, European regulators tend to err on the side of caution.

For example, you may remember when I wrote about five food additives they’ve banned across the pond and beyond, but are still being fed to us in America — chemicals linked to cancer, endocrine disruption and type 2 diabetes.

Well, it looks like the U.S. Food and Drug Administration (FDA) is finally coming around on at least one of those additives found in drinks enjoyed by almost 65 percent of Americans daily — and linked to thyroid toxicity…

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The additive that makes sodas toxic

In November 2023, the FDA proposed a measure that would ban the use of brominated vegetable oil (BVO) in the U.S.

Used to keep the citrus flavoring in beverages from separating, until now the FDA has considered its use in small quantities as safe.

But the agency was prompted to revisit this safety classification after scientific evidence from toxicology studies conducted in collaboration with the National Institutes of Health (NIH) found that an accumulation of BVO is toxic to the thyroid.

Your thyroid produces hormones that are instrumental in helping control blood pressure, body temperature, metabolism and response to other hormones. Disrupting the function of the thyroid means throwing these key processes out of whack.

And that’s not all. Brominated vegetable oil has also been linked with skin and mucous membrane irritation, fatigue, loss of muscle coordination and memory problems.

BVO was given the “Generally Recognized as Safe” (GRAS) designation by the FDA in the 1970s. However, as a result of these recent findings, the FDA now deems the continued use of BVO in food as unsafe.

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Some beverage manufacturers jumped ahead of the game to remove BVO from product formulations a few years ago when questions first began to arise about its safety. One prominent example is PepsiCo, which removed BVO from its citrus-flavored Gatorade in 2013 because consumers perceived the product in a negative light.

But according to the Environmental Working Group (EWG), up to at least 90 products common on your grocer’s shelves contain BVO — mostly sodas, sports drinks and juice-like drinks with a citrusy flavor, including regional and store brands. You can view EWG’s complete list here.

The state of California has already made moves to ban the use of BVO and three other food ingredients within its borders: potassium bromate, propylparaben and FD&C Red No. 3, or red dye No. 3. By 2027, manufacturers will no longer be able to use these four ingredients in food sold in California.

The FDA says it continues to assess the safety of various chemicals in food based on the latest scientific findings and legal requirements, including the recent California law. In fact, the agency is in the process of reviewing FD&C Red No. 3 and says a decision on the chemical is forthcoming.

Going forward, the FDA plans to streamline the process of evaluating chemicals in the food supply by creating an “Office of Food Chemical Safety, Dietary Supplements, and Innovation.” This office is part of the agency’s proposed Human Foods Program (HFP) transformation to enhance its review of food chemical safety.

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Dodging brominated vegetable oil

While the FDA’s announcement about BVO is good news, it could take a while for its proposed ban to take effect.

Until then, you’ll want to check your beverage labels to make sure BVO isn’t an ingredient.

Of course, one surefire way to reduce the danger of accidentally ingesting BVO is to ditch sodas —and it’s far from the only reason you should…

A large study found that postmenopausal women who drank one or more sweetened beverages per day were 78 percent more likely to develop liver cancer, compared with women who didn’t (or consumed less than three servings per month). The researchers also concluded that even consuming just one a day increased the likelihood of liver cancer by 73 percent!

And of course, aspartame, the most commonly used artificial sweetener in sodas has been listed as “possibly carcinogenic to humans” by the International Agency for Research on Cancer, the cancer research arm of the World Health Organization.

These reasons add up to why sweetened beverages are considered one of the two worst ultraprocessed foods for our health.

It looks like giving them up altogether is the wise thing to do.

Sources:

FDA moves to ban drink additive linked to thyroid issues, memory loss — UPI News

California bans four food additives linked to health issues — UPI

FDA Proposes to Ban Food Additive, Continues Assessments of Additional Chemicals — U.S. Food and Drug Administration

Why Is Red Dye No. 3 Banned in Cosmetics but Still Allowed in Food? — Consumer Reports

The post The thyroid-poisoning additive in popular drinks appeared first on Easy Health Options®.

Many of us strive to follow a healthy diet, exercise regularly, reach for vitamins and supplements that support healthier aging, and may even try calorie restriction in hopes of that end goal.

Those with deep pockets can employ extreme (and expensive) “hacks” like hyperbaric oxygen chamber treatments, cryogenic chamber sessions, red light therapy, stem cell treatments and personal doctors or life coaches that keep them on strict regimens.

But what if living a longer, healthier life was as simple as popping a pill? Sounds like a pipe dream, right?

Well, we may be inching closer to that reality just by using currently approved drugs…

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A longevity drug cocktail?

A research team led by scientists from the Max Planck Institute in Germany sought to explore the impact of a pair of anticancer drugs, rapamycin and trametinib, on the lifespan of mice.

Rapamycin, which is also used to prevent organ rejection, has shown promise in extending lifespan in previous animal studies. Trametinib has been shown to extend the lifespan of fruit flies.

The researchers administered regular doses of rapamycin, trametinib, or both to hundreds of mice starting at six months of age. They then measured their survival for the remainder of their lives.

Each drug showed potential. Rapamycin alone extended the lifespan of mice by 17% to 18%. At the same time, trametinib showed a more modest boost in longevity, at 7% to 16%.

But it was together that the drugs truly shone…

Mice treated with a combination of rapamycin and trametinib saw a lifespan extension of about 26% to 35%.

Both of the drugs act on the same signaling pathway, known as the Ras/Insulin/TOR network, which plays a central role in aging. However, they target different points along the path.

When the researchers analyzed gene expression, they found specific changes that occurred only when the drugs were administered together.

What’s more, the combo treatment delayed the growth of liver and spleen tumors in the mice and reduced chronic inflammation in the brain. The treated mice also seemed more active in older age than the control mice, weighed less and experienced a slower decline in heart function.

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But we’re not there yet

While these results are encouraging, they are far from proving that these drugs are a miracle longevity elixir.

They need to be tested in humans first, and the researchers agree that they likely won’t have the same impact on lifespan as they did in the mice, but could hold benefits for our golden years.

“While we do not expect a similar extension to human lifespans as we found in mice, we hope that the drugs we’re investigating could help people to stay healthy and disease-free for longer late in life,” says geneticist Linda Partridge, co-senior author of the study.

“Further research in humans in years to come will help us to elucidate how these drugs may be useful to people, and who might be able to benefit,” Partridge adds.

Since both rapamycin and trametinib are already approved for use in humans in the US and European Union, human trials for the drug combo could begin soon.

But I can’t help but wonder what the cost of these cancer drugs will be if they’re approved for off-label use, and if we are looking at just another health hack for the rich and famous. It’s doubtful insurance companies will foot the bill in the absence of a medical diagnosis.

In the meantime, don’t sit idly by while your longevity clock keeps ticking away. Check out how a simple vitamin slowed biological age by three years— and in human subjects! Plus, 10 micronutrients that turn the key on age reversal.

Sources:

Live longer — EurekAlert!

Anti-Aging Cocktail Extends Mouse Lifespan by About 30 Percent — Science Alert

The geroprotectors trametinib and rapamycin combine additively to extend mouse healthspan and lifespan — Nature Aging

Hyperbaric oxygen therapy for healthy aging: From mechanisms to therapeutics — Redox  Biology

Cold and longevity: Can cold exposure counteract aging? — Life Sciences

Near-infrared light increases ATP, extends lifespan and improves mobility in aged Drosophila melanogaster— Biology Letters

Anti-aging based on stem cell therapy: A scoping review — World Journal of Experimental Medicine

The post Two existing drugs that could extend human lifespan appeared first on Easy Health Options®.

The opposite — a sedentary lifestyle — has been linked to everything from heart failure and stroke to dementia and more.

But let’s face it, 10,000 is a big number.

And fitting that many steps into every day can be a challenge.

Luckily, research shows that while getting close to 10,000 steps a day might be an admirable goal, it takes far fewer steps to tip the scales in favor of a healthy heart and longevity.

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Surprisingly lower amount of steps benefits the heart

Researchers delved into almost seven years’ worth of data from 72,174 volunteers in the UK Biobank. Each participant had worn wrist accelerometers to track their average number of steps and time spent sitting for seven days.

As expected, the results confirmed between 9,000 and 10,000 steps daily to be the perfect amount to shoot for to counteract a highly sedentary lifestyle (sitting for over 10.6 hours daily). In fact, getting this high number of steps in daily lowered heart disease risk by 21 percent and overall mortality risk by almost 40 percent.

However, the researchers also found that regardless of a participant’s sedentary time, 50 percent of those benefits kicked in at around 4,000 to 4,500 daily steps.

And the data showed that getting any amount of steps above 2,200 per day led to lower heart disease and death risk whether a person spent only a little bit of time each day sedentary or landed in that high sedentary lifestyle group.

This means that if you can’t get to 10,000, you can still experience significant benefits from a much lower level of walking time.

And that’s good news because, on average, getting in 2,200 steps will likely take only about 20 minutes.

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More benefits from walking

So if reaching 10,000 steps in a day is just more than you can handle, get in the amount that works for you — but try for somewhere between 2,200 and 4,500, at least — as long as you try to do it every day. But the more, the better — if you can work up to it.

If you just need a little help in the “get-up-and-go” department, try dimethylglycine, or DMG for short. Since the 1970’s, it’s been used by athletes looking for a performance edge, but the supplement has been found to safely support heart and immune system health and fight inflammation.

Besides supporting your heart and increasing your chances for a longer lifespan, walking offers too much to pass up. See what else a little walking every day can do for you:

• Living with osteoarthritis of your knees? Walking could help you avoid knee replacement surgery;
• Walking can help you walk away from low back pain;
• Walking also boosts metabolism, which can help you stay fit and trim;
• It can also help beat back pain and protect against breast cancer;
• Increase your walking pace to lower the risk of Afib by 46%!

Sources:

Scientists Reveal The Optimal Number of Daily Steps to Offset Sitting Down – Science Alert

The post The least number of steps to lower heart disease risk appeared first on Easy Health Options®.

And if your numbers weren’t just right, you may have been prescribed statins. Your doctor may have even warned about muscle pain, a well-known side effect of the drug. But that’s not the complete picture…

The list of side effects has grown to include everything from an increased risk of diabetes and cataracts to accelerated onset of Parkinson’s and double the risk for dementia.

Unfortunately, the guidelines for prescribing statins aren’t so cut and dry, as few cardiologists will tell you, but they’re also not the only answer for tackling cholesterol…

There’s an ancient seed that could offer significant improvements in cholesterol without that laundry list of side effects.

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The history of Nigella sativa

The Nigella sativa plant, famous for its black seed, grows on a small shrub with green leaves and white and purplish flowers.

Although you can find the plant growing worldwide, its origins are believed to be in North Africa and Asia.

In fact, Egyptian Kings and Queens treasured the oil pressed from the seed as a remedy that offered whole-body protection, calling it the Pharaoh’s Oil. History shows it was so popular it was mentioned in the bible, and King Tut wanted to be buried with it for use in the afterlife.

Black seed, also called black cumin seed, is treasured thanks to its bioactive components, especially thymoquinone and thymohydroquinone.

Because of the therapeutic potential of these natural actives, studies have been done on Nigella sativa to see how it can improve blood sugar, weight, immune function, and liver and kidney protection. It has also been investigated as a way to reduce spasms, relieve pain and fight off germs and infections — and much more.

And, of course, researchers have been highly interested in the seed’s effect on cholesterol.

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Study after study shows cholesterol benefits

So what have they found?

Let’s break down the research…

One study asked sedentary overweight women to take either a placebo or 2,000 mg of Nigella sativa for eight weeks while also participating in an aerobics training program. 

The results showed the group supplementing Nigella sativa experienced:

• A five percent increase in HDL levels (good cholesterol)
• A decrease in total cholesterol levels of five percent or greater
• A reduction in LDL cholesterol of up to seven percent (the bad cholesterol)
• An eight to 16 percent reduction in triglyceride levels

Additional studies have demonstrated that:

• In people with metabolic syndrome, black seed oil significantly elevated blood HDL and lowered LDL.
• In patients with high cholesterol, 1,000 mg of the oil per day for two months before breakfast lowered total cholesterol, LDL and triglycerides while increasing HDL.
• When comparing statin use versus Nigella sativa plus statins in people with acute coronary syndrome, total cholesterol, LDL and triglycerides were considerably lower when adding in the oil than in the statin alone group.
• In men with cholesterol problems and high blood pressure taking either a placebo or 100/200 mg of black seed oil two times daily, researchers found nigella sativa improved all cholesterol numbers. They saw more improvements as the dose went up.

That’s a lot of research pointing to one thing…

Nigella sativa or black seed oil could be a valuable aid for improving cholesterol naturally.

Even though the seed is a surprisingly popular Mediterranean spice, it’s also available in supplement form.

Sources:

The Effects of 8-week Nigella sativa Supplementation and Aerobic Training on Lipid Profile and VO2 max in Sedentary Overweight Females  — NCBI

A review on hypo-cholesterolemic activity of Nigella sativa seeds and its extracts — NIH

Using Black Seed to Lower Cholesterol — Verywell health

The post The seed that takes down bad cholesterol and raises good appeared first on Easy Health Options®.

Such a fountain may exist only in legend. But science hasn’t stopped searching for ways to stop the clock…

They’ve zeroed in on a popular vitamin that’s demonstrating much promise in the real fight against aging.

Not only does it appear to slow biological aging, but it also reduces risk for diseases generally associated with the aging process…

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The nutrient that can help protect telomeres

Telomeres are repeating sequences of DNA at the ends of chromosomes that keep the chromosomes from deteriorating. They’re sort of like the plastic end caps that prevent the fibers that make up a rope or shoelace from unraveling.

As we grow older, these telomeres become shorter. And that exposes our chromosomes to degradation that increases risks for various age-related diseases.

So far, investigators have explored two nutrients for their potential in supporting telomere length: vitamin D and omega-3 fatty acids. But those studies have been short-term and small in scale, and results have been inconsistent.

This prompted researchers led by Mass General Brigham and the Medical College of Georgia, Augusta University to conduct a sub-study of the VITamin D and OmegA-3 TriaL (VITAL) trial. This randomized, double-blind, placebo-controlled trial tracked supplementation of these two nutrients in U.S. females aged 55 and older and males aged 50 and older for five years.

The VITAL Telomere sub-study included 1,054 of these participants, whose telomere length in white blood cells was assessed at the start of the study and again at years two and four. And when it came to telomer length, vitamin D3 took center stage…

Compared to those on a placebo, individuals taking 2,000 IU/day of vitamin D3 experienced significantly reduced telomere shortening over a four-year period.

In fact, this reduction prevented the equivalent of nearly three years of aging! The impact on age-related disease was also impactful. Daily, vitamin D3 supplementation:

• Reduced the incidence of advanced (metastatic or fatal) cancer by 17%.
• Reduced all incident autoimmune diseases by 22%.
• Decreased circulating high sensitivity-C-reactive protein (a chronic disease biomarker) concentrations by 19%.

“VITAL is the first large-scale and long-term randomized trial to show that vitamin D supplements protect telomeres and preserve telomere length,” says co-author Dr. JoAnn Manson, principal investigator of VITAL and chief of the Division of Preventive Medicine at Brigham and Women’s Hospital, a founding member of the Mass General Brigham healthcare system.

“Our findings suggest that targeted vitamin D supplementation may be a promising strategy to counter a biological aging process, although further research is warranted,” says Dr. Haidong Zhu, first author of the report and a molecular geneticist at the Medical College of Georgia.

By contrast, daily supplementation with one gram of omega-3 fatty acids demonstrated no significant impact on telomere length throughout the follow-up.

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Don’t skimp on your vitamin D3

Unfortunately, the best source of vitamin D — sun exposure — is a double-edged sword, as it can cause skin cancer and visible aging of the skin. Wearing sunscreen to protect yourself from the sun’s ill effects also prevents the sun from triggering your body’s natural vitamin D production.

So, how about diet? There are only a few foods that contain vitamin D3 (the best form of vitamin D), including beef liver, egg yolks and cheese. And you would have to eat a ton of these foods to get the recommended daily amount of the nutrient.

That’s why supplementation is the best way to ensure you’re getting enough vitamin D3. Make sure you’re getting at least 2,000 IUs a day (the amount used in the VITAL study), but if you’re vitamin D-deficient, you’ll want to up that to a therapeutic dosage of 4,000 to 5,000 IUs daily, at least. The benefits are just too good to pass up…

Other health benefits associated with vitamin D include:

• A 20% reduction in the risk factors of heart disease
• Improved mood
• Increased insulin sensitivity
• Decreased formation of insoluble proteins such as beta amyloid

Sources:

Vitamin D supplements show signs of protection against biological aging — ScienceDaily

Vitamin D₃ and marine ω-3 fatty acids supplementation and leukocyte telomere length: 4-year findings from the VITamin D and OmegA-3 TriaL (VITAL) randomized controlled trial — The American Journal of Clinical Nutrition

The post Slow biological aging 3 years with a vitamin appeared first on Easy Health Options®.

But what happens if you become hard of hearing?

Your friend must knock harder for you to hear them at the door.

This is kind of what happens to your body as you develop insulin resistance.

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What is insulin resistance?

Insulin is a hormone secreted by the pancreas that is crucial for converting food into energy or storing that energy for later use.

When your blood glucose levels rise after a meal, the pancreas responds by producing insulin. The insulin, in turn, helps sugar enter your cells so that the amount of sugar in your bloodstream falls back to a normal range.

The “knock at the door” is insulin telling your insulin-sensitive muscle, liver and fat cells that glucose needs to get in.

But changes in the body can cause those cells to become hard of hearing. They don’t respond as effectively as they should to take up or store glucose from your bloodstream.

What does the pancreas do? It produces even more insulin.

Your blood sugar level might be normal, but you need way more insulin to get it into a normal range because the cells have lost their sensitivity. At this point, you are insulin-resistant.

Eventually, the pancreas can’t make enough insulin to knock as loudly as it needs to, and blood sugar levels start to rise. At this point, if things are not corrected, prediabetes, then type 2 diabetes could be around the corner.

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Who is at risk for insulin resistance?

Genes and older age can make us more prone to insulin resistance, but two lifestyle factors — excess belly fat and physical inactivity — are considered the two main contributing factors.

Other factors that may increase the risk for insulin resistance include:

• A diet high in processed, high-carbohydrate and high-fat foods requires more insulin. These foods tend to be high glycemic index foods.
• A family history of type 2 diabetes.
• Having high triglycerides, high LDL cholesterol or low HDL cholesterol.

Some hormonal conditions are associated with insulin resistance, including:

Cushing’s syndrome: Cortisol, best known as the stress hormone, is necessary in the conversion of blood sugar to energy. However excess cortisol (which can occur due to chronic stress or Adrenal fatigue) can counteract insulin and cause insulin resistance.

Hypothyroidism (or low thyroid): The thyroid plays a big role in regulating your metabolism. When it doesn’t produce enough thyroid hormone, it slows glucose metabolism which can lead to insulin resistance.

Polycystic ovary syndrome (PCOS): People with PCOS are more likely to be overweight and have metabolic syndrome. That sets a great many of them up for insulin resistance.

Hypertension, heart disease and nonalcoholic fatty liver disease (NAFLD) are also linked to insulin resistance.

Some drugs can set you up for insulin resistance, including glucocorticoids (prednisone is one), some antipsychotics and some HIV medicines.

What are the early signs of insulin resistance?

• A waistline over 40 inches in men and 35 inches in women
• Blood pressure readings of 130/80 or higher
• A fasting glucose level over 100 mg/dL
• A fasting triglyceride level over 150 mg/dL
• A HDL cholesterol level under 40 mg/dL in men and 50 mg/dL in women
• Skin tags
• Patches of dark, velvety skin called acanthosis nigricans
• Eye changes that can lead to diabetes-related retinopathy
• Feeling hungry or tired all the time
• Extreme thirst
• Irregular menstrual periods

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Get your insulin sensitivity back

Make no mistake: insulin resistance is a sign that your metabolism is seriously out of whack.

If your pancreas is showing signs of exhaustion, the sooner you start an all-out effort to get your metabolism back in line, the better. Starting here:

Lose a few pounds. Excess weight, especially belly fat, tops the list of causes of insulin resistance. If you need help finding the energy to exercise, enlist the help of the antioxidant, PQQ, found to go after visceral fat while delivering the energy the body needs to exercise.

Exercise more. Many studies have also found that resistance training can increase insulin sensitivity among men and women with or without diabetes.

Eat healthy foods.  Add more soluble fiber and colorful fruits (like berries) and vegetables to your diet. The more colorful the better, as these contain antioxidants that have been shown to help with weight loss and curb insulin resistance. Cut back on high glycemic foods like carbs (which stimulate excess insulin production), sugars and processed starches. Also, eat much less red meat.

Keep your vitamin D levels healthy. Several studies have connected low vitamin D levels to insulin sensitivity.

Boost CoQ10 levels. Research has also shown that low CoQ10 levels correlate to insulin resistance.

Get more sleep. Give up night owl habits, like staying up late and getting less sleep. Getting more sleep can help reverse its effects.

Reduce stress. Ongoing stress keeps your cortisol level high, which stimulates the breakdown of nutrients and increases blood sugar.

Sources:

What Is Insulin Resistance and How Do You Know if You Have It? — NY Times

Insulin Resistance — Cleveland Clinic

The post Insulin resistance: Early signs and ditching the diabetes danger appeared first on Easy Health Options®.

Some believe genetic makeup is a factor, but the truth is, in less than one percent of people, Alzheimer’s is caused by specific genetic changes that almost guarantee they will develop the disease.

For the rest of us, the answer seems to lie in a combination of genetics, other health conditions and lifestyle and environmental risk factors that impact the brain over time — including sleep patterns, lack of exercise, diet, metabolic syndrome, type 2 diabetes, depression, alcohol and air pollution, to name a few.

But there is one lifestyle-related element that scientists are just beginning to focus on: stress.

A previous study found that having chronic stress or depression more than doubled the risk of Alzheimer’s. And in those who had both chronic stress and depression, the Alzheimer’s risk was four times as high!

This is only one study. But there is more research that indicates the stress connection should be taken seriously — especially for women…

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The role of cortisol in Alzheimer’s

A team of researchers led by The University of Texas Health Science Center at San Antonio (UT Health San Antonio) notes that so-called “sporadic” Alzheimer’s is the leading cause of cognitive decline in older adults. During this prolonged asymptomatic phase, amyloid beta accumulates into amyloid plaques, ultimately leading to progressive cognitive decline.

Unfortunately, these biological changes are already well-established by the time symptoms appear, which is why effective early interventions are needed.

Some investigators have zeroed in on cortisol, also known as the “stress hormone.” Cortisol is necessary for maintaining cellular balance and triggering the stress response, also known as the fight-or-flight response.

But when the release of cortisol in the body becomes chronically elevated, it can cause all kinds of problems.

Some studies have already indicated that higher blood cortisol levels are linked to an increased likelihood of developing Alzheimer’s.

Stress, Alzheimer’s and post-menopausal women

To shed further light on these findings, researchers from the University of Texas Health San Antonio led a comparison of cortisol levels in 305 cognitively unimpaired, middle-aged participants from the Framingham Heart Study. A little under half of the participants were women.

Approximately 15 years later, they assessed the burden of amyloid beta and tau protein using PET scans. This allowed them to investigate cortisol’s impact at an earlier stage in Alzheimer’s development, when interventions might be most effective.

The findings were sobering. The researchers discovered high levels of cortisol in midlife were linked to increased deposits of amyloid in post-menopausal women. No significant associations were observed in men.

The researchers theorized that post-menopausal hormone changes may amplify cortisol’s effects on amyloid buildup. Both estrogen and testosterone have neuroprotective effects that might mitigate cortisol’s negative impact on neural tissues, but levels of both hormones drop dramatically in menopause. This indicates that both stress reduction and hormonal interventions may help prevent Alzheimer’s in at-risk women.

Dr. Arash Salardini, a professor at UT Health San Antonio and first author of the study, adds that follow-up of the study cohort is necessary to determine whether these early amyloid changes translate into clinical symptoms, as well as to clarify the causal role of cortisol in Alzheimer’s development.

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Women and their higher burden of Alzheimer’s

None of this is really surprising. More than two-thirds of people living with Alzheimer’s are women, and hormones have been indicated previously.

But understanding the involvement of yet another hormone, cortisol, gives us one more target to focus on to reduce the odds.

If you’re a woman and you find yourself constantly stressed out, talk to your doctor about testing your cortisol levels. Certain medical conditions, including Cushing’s syndrome, adrenal fatigue, adrenal tumors and pituitary tumors, can cause chronically elevated cortisol levels.

Depression and anxiety have been linked to elevated cortisol levels due to the disruption of the hypothalamic-pituitary-adrenal (HPA) axis, which regulates cortisol production. 

 If outside stressors are the problem, lifestyle interventions, like these, may help:

• Yoga. With its emphasis on slow, deliberate movement and breathing, yoga not only helps mellow you out but has also been shown to help build up the brain’s left prefrontal cortex, an area responsible for critical mental functions such as learning and memory.
• Meditation. Research has shown that practicing meditation regularly can increase brain density, boost connections between neurons, decrease symptoms of depression and anxiety, provide clarity of thought and increase positive mood endorphins.
• Emotional regulation. By purposely changing your focus and heading off negative emotions and stress before they arise, you can reduce ruminating — which is linked to cognitive decline and brain aging.
• L-theanine. A 250 to 400 mg dose of this amino acid daily can help lower your cortisol levels and sharpen your focus and attention.

Sources:

Stress can lead to Alzheimer’s disease in women who are post-menopausal, UT Health San Antonio study finds — EurekAlert!

Elevated serum cortisol associated with early-detected increase of brain amyloid deposition in Alzheimer’s disease imaging biomarkers among menopausal women: The Framingham Heart Study — Alzheimer’s & Dementia

Alzheimer’s disease — Mayo Clinic

The post How stress becomes a post-menopausal Alzheimer’s trigger appeared first on Easy Health Options®.

If you’re one of those people sticking to a no-carb, low-carb, keto or Atkins-inspired diet, prepare to have your mind changed.

In a lot of people’s minds, carbs fall into the category of “they’re fattening, and they don’t hold much nutritional value,” and they’re bad for you.

Well, nothing could be further from the truth.

And if you’re a woman, eating the right carbs during middle age can be like an insurance policy that will all but guarantee your health in old age…

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Quality carbs in midlife linked to a healthier old age

You probably know that there are two kinds of carbohydrates: refined and unrefined.

Refined carbs are mostly white. Think white bread and white rice. But they also include pastries, waffles, pizza dough, and muffins. Refined carbs are considered ultra-processed foods.

Unrefined carbs are high-quality carbs from natural, whole foods that haven’t undergone significant processing, so they retain their natural fiber and nutrients, like whole grains (such as brown rice, quinoa, and oatmeal), fruits, vegetables, legumes, nuts and seeds.

Researchers from Tufts University and the Harvard T.H. Chan School of Public Health sought to determine the long-term significance of our carb choices, not just in the present — but thirty years down the road.

“We’ve all heard that different carbohydrates can affect health differently, whether for weight, energy or blood sugar levels. But rather than just look at the immediate effects of these macronutrients, we wanted to understand what they might mean for good health 30 years later,” says Andres Ardisson Korat, the study’s lead author.

The researchers analyzed data from the Nurses’ Health Study collected every four years between 1984 and 2016 from 47,000 women aged 70 to 93 years in 2016, including specifics about their intakes of carbohydrates and dietary fiber.

Dietary glycemic index and glycemic load were also derived from the validated questionnaires.

Parameters set to define healthy aging included the absence of 11 major chronic diseases, lack of cognitive and physical function impairments, and good mental health, as self-reported in questionnaires.

A total of 3,706 participants met the definition of healthy aging. How did carbs play into all the data?

It turns out that carb quality intake in midlife is a strong predictor of healthy aging:

• Higher intakes of total carbs, high-quality carbs from fruits, vegetables and legumes and total dietary fiber were linked to between six and 37% greater likelihood of healthy aging,
• On the flip side of the coin, intakes of refined carbs (added sugars, white grains and potatoes) were associated with 13% lower odds of healthy aging.

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How to choose your carbs wisely

Carbohydrates are a vital component of a healthy diet.

The Dietary Guidelines for Americans recommend that carbohydrates make up 45% to 65% of total daily calories. So, if you’re eating 2,000 calories a day, between 900 and 1,300 calories should be from carbohydrates.

If you still need tips on choosing your carbs wisely, here’s some advice from the Mayo Clinic:

• Focus on eating fiber-rich fruits and vegetables.
• Replace refined grains with whole grains.
• Eat more beans, peas, and lentils.
• Stick to low-fat dairy products, which limit saturated fat, but be mindful of added sugar.
• Limit added sugars from any source; the Dietary Guidelines for Americans recommend getting less than 10% of your daily calories from added sugar.

And about those ultra-processed foods that can contain refined carbs — avoid them like the plague. They can send aging into overdrive.

The bottom line: what’s on your plate today can help you or haunt you. Make good carb choices for healthy aging.

Sources:

In healthy aging, carb quality counts — Eureka Alert

Dietary Carbohydrate Intake, Carbohydrate Quality, and Healthy Aging in Women — JAMA Network Open

Carbohydrates: How carbs fit into a healthy diet — Mayo Clinic

Dietary guidelines for Americans 2020-2025 — US Department of Agriculture

The post Don’t give up these carbs, for healthiest aging appeared first on Easy Health Options®.